نتایج جستجو برای: van laere syndrome

تعداد نتایج: 686720  

2017
Venkatraman Thulasi Aravindhan Veerapandiyan Beth A. Pletcher Chun M. Tong Xue Ming

Brown-Vialetto-Van Laere syndrome is a rare disorder characterized by motor, sensory, and cranial neuronopathies, associated with mutations in SLC52A2 and SLC52A3 genes that code for human riboflavin transporters RFVT2 and RFVT3, respectively. The authors describe the clinical course of a 6-year-old girl with Brown-Vialetto-Van Laere syndrome and a novel homozygous mutation c.1156T>C in the SLC...

Journal: :Orphanet Journal of Rare Diseases 2008
Sivakumar Sathasivam

The Brown-Vialetto-Van Laere syndrome (BVVL) is a rare neurological disorder characterized by progressive pontobulbar palsy associated with sensorineural deafness. Fifty-eight cases have been reported in just over 100 years. The female to male ratio is approximately 3:1. The age of onset of the initial symptom varies from infancy to the third decade. The syndrome most frequently presents with s...

Journal: :iranian journal of neurology 0
samira yadegari m.d, department of neurology, tehran university of medical sciences, tehran, iran. askar ghorbani m.d, department of neurology, tehran university of medical sciences, tehran, iran mitra ansari dezfouli school of biology, university college of science, university of tehran, tehran, iran shahriar nafissi m.d, department of neurology, tehran university of medical sciences, tehran, iran

brown-vialetto-van laere syndrome (bvvls) is a rare neurological disorder. we report our finding about four patients clinically and electrophysiologically diagnosed as bvvls and denoted their clinical features with comparison to previous reports. the first symptom was bilateral hearing loss and the onset of other cranial nerves involvement varied between 0-15 years. our patients represented som...

Journal: :Brain : a journal of neurology 2012
Janel O Johnson J Raphael Gibbs Andre Megarbane J Andoni Urtizberea Dena G Hernandez A Reghan Foley Sampath Arepalli Amelie Pandraud Javier Simón-Sánchez Peter Clayton Mary M Reilly Francesco Muntoni Yevgeniya Abramzon Henry Houlden Andrew B Singleton

Brown-Vialetto-Van Laere syndrome was first described in 1894 as a rare neurodegenerative disorder characterized by progressive sensorineural deafness in combination with childhood amyotrophic lateral sclerosis. Mutations in the gene, SLC52A3 (formerly C20orf54), one of three known riboflavin transporter genes, have recently been shown to underlie a number of severe cases of Brown-Vialetto-Van ...

Journal: :Orphanet Journal of Rare Diseases 2008

2017
Andreea Manole Zane Jaunmuktane Iain Hargreaves Marthe H R Ludtmann Vincenzo Salpietro Oscar D Bello Simon Pope Amelie Pandraud Alejandro Horga Renata S Scalco Abi Li Balasubramaniem Ashokkumar Charles M Lourenço Simon Heales Rita Horvath Patrick F Chinnery Camilo Toro Andrew B Singleton Thomas S Jacques Andrey Y Abramov Francesco Muntoni Michael G Hanna Mary M Reilly Tamas Revesz Dimitri M Kullmann James E C Jepson Henry Houlden

Brown-Vialetto-Van Laere syndrome represents a phenotypic spectrum of motor, sensory, and cranial nerve neuropathy, often with ataxia, optic atrophy and respiratory problems leading to ventilator-dependence. Loss-of-function mutations in two riboflavin transporter genes, SLC52A2 and SLC52A3, have recently been linked to Brown-Vialetto-Van Laere syndrome. However, the genetic frequency, neuropat...

Journal: :Indian pediatrics 1996
V Puri A Rohtagi P S Parihar

The syndrome described by Brown(l) Vialetto(2) and Van Laere(3) is a rare, often familial, characterized by bilateral nerve deafness followed or accompanied by involvement of various motor cranial nerves, e.g., 7th and 9th to 12th, rarely 3rd, 5th and 6th. On rare occasions, there may also be involvement of spinal motor nerves and less commonly of upper motor neurons. The onset of the disease i...

Journal: :Pediatric Neurology Briefs 2014

Journal: :Journal of medical genetics 1990
S A Hawkins N C Nevin A E Harding

A female with the Brown-Vialetto-Van Laere syndrome is described. The patient's father, a paternal uncle, and possibly a paternal first cousin had neurosensory deafness and a paternal aunt had clinical symptoms indicative of the syndrome. This family raises the possibility that the disorder is genetically heterogeneous with autosomal recessive and autosomal dominant forms. Alternatively, it cou...

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