نتایج جستجو برای: q23

تعداد نتایج: 826  

2003
Chien-Shing Chen H. B. Sorensen Peter H. Domer Gregory H. Reaman Stanley J. Korsmeyer Nyla A. Heerema G. Denman Hammond John H. Kersey

Acute lymphoblastic leukemia (ALL) in infants generally shows distinctive biologic features and has a poor prognosis. Cytogenetic studies indicate that many infant leukemias have chromosome 1 1 q23 translocations. Because of these findings and the distinct clinical features of infant leukemia, we investigated 30 cases of infant ALL for molecular defects of l l q23. Fourteen cases had cytogeneti...

2002
F. G. Behm F. O. Smith

Monoclonal antibody 7.1, which recognizes the chondroitin sulfate proteoglycan molecule NG2, was used to screen prospectively blast cells from 104 consecutive children at initial presentation with acute lymphoblastic leukemia (ALL). Reactivity with this antibody was found in 9 cases (8.6’701, of whom5hadat(4;11)(q21;q23)and4hadat(11;19)(p13;q23). None of the NG2cases had either translocation. S...

2003
Stephen P. Hunger Douglas C. Tkachuk

Chromosome band 1 1 q23 is a site of recurrent translocations and interstitial deletions in human leukemias. Recent studies have shown that the 1 1 q23 gene HRX is fused to heterologous genes from chromosomes 4 or 19 after t(4;l l)(q21 ;q23) and t(ll;19)(q23; p l3 ) translocations to create fusion genes encoding proteins with structural features of chimeric transcription factors. In this report...

Journal: :Blood 2011
Jenny E Kuipers Eva A Coenen Brian V Balgobind Jan Stary Andre Baruchel Valerie de Haas Eveline S J M de Bont Dirk Reinhardt Gertjan J L Kaspers Jacqueline Cloos Astrid A Danen-van Oorschot Monique L den Boer Rolf Marschalek Claus Meyer Rob Pieters C Michel Zwaan Marry M van den Heuvel-Eibrink

Pediatric mixed-lineage leukemia (MLL)-rearranged acute monoblastic leukemia with t(9;11)(p22;q23) has a favorable outcome compared with other MLL-rearranged AML. The biologic background for this difference remains unknown. Therefore, we compared gene expression profiles (GEPs; Affymetrix HGU133 + 2.0) of 26 t(9;11)(p22;q23) patients with 42 other MLL-rearranged AML patients to identify differe...

Journal: :Blood 2009
Brian V Balgobind Susana C Raimondi Jochen Harbott Martin Zimmermann Todd A Alonzo Anne Auvrignon H Berna Beverloo Myron Chang Ursula Creutzig Michael N Dworzak Erik Forestier Brenda Gibson Henrik Hasle Christine J Harrison Nyla A Heerema Gertjan J L Kaspers Anna Leszl Nathalia Litvinko Luca Lo Nigro Akira Morimoto Christine Perot Rob Pieters Dirk Reinhardt Jeffrey E Rubnitz Franklin O Smith Jan Stary Irina Stasevich Sabine Strehl Takashi Taga Daisuke Tomizawa David Webb Zuzana Zemanova C Michel Zwaan Marry M van den Heuvel-Eibrink

Translocations involving chromosome 11q23 frequently occur in pediatric acute myeloid leukemia (AML) and are associated with poor prognosis. In most cases, the MLL gene is involved, and more than 50 translocation partners have been described. Clinical outcome data of the 11q23-rearranged subgroups are scarce because most 11q23 series are too small for meaningful analysis of subgroups, although ...

2003
Michael J. Thirman Richard A. Larson Michelle M. Le Beau Jens Pedersen-Bjergaard Janet D. Rowley

Chromosome band 1 1 q23 is frequently involved in acute myeloid leukemia (AML) and acute lymphoblastic leukemia (ALL) de novo, as well as in myelodysplastic syndromes (MDS) and lymphoma. Five percent to 15% of patients treated with chemotherapy for a primary neoplasm develop therapy-related AML (t-AML) that may show rearrangements, usually translocations involving band 1 1 q23 or, less often, 2...

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2013

Journal: :Cancer research 2001
F Shirasaki M Takata N Hatta K Takehara

KiSS1 is a putative melanoma metastasis suppressor gene, the expression of which may be regulated by another gene(s) mapping to chromosome 6q16.3-q23. To additionally elucidate the role of KiSS1 in the progression of human melanoma in vivo, we examined KiSS1 mRNA expression in 51 melanocytic tumors with various stages of progression by in situ hybridization. We also examined a correlation betwe...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1990
J D Rowley M O Diaz R Espinosa Y D Patel E van Melle S Ziemin P Taillon-Miller P Lichter G A Evans J H Kersey

Translocations involving chromosome 11, band q23, are frequent recurring abnormalities in human acute lymphoblastic and acute myeloid leukemia. We used 19 biotin-labeled probes derived from genes and anonymous cosmids for hybridization to metaphase chromosomes from leukemia cells that contained four translocations involving band 11q23: t(4;11)(q21;q23), t(6;11)(q27;q23), t(9;11)(p22;q23), and t...

2011
Ayse Elif Erson Elizabeth M Petty

Hugo: TRIM37 Other names: MUL; KIAA0898; POB1; TEF3 Location: 17q23.2 Local order: Genes flanking TRIM37 oriented from centromere to telomere on 17q23 are: RAD51C, 17q22-q23, D51 homolog C (S. Cerevisiae) PPM1E, 17q23.2, protein phosphatase 1E (PP2C domain containing) TRIM37, 17q22-q23, tripartite motif-containing 37 FAM33A 17q23.2, family with sequence similarity 33, member A PRR11(FLJ11029) 1...

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