نتایج جستجو برای: nuclear hypertrophy

تعداد نتایج: 279538  

2013
Akira Funayama Tetsuro Shishido Shunsuke Netsu Taro Narumi Shinpei Kadowaki Hiroki Takahashi Takuya Miyamoto Tetsu Watanabe Chang-Hoon Woo Jun-ichi Abe Koichiro Kuwahara Kazuwa Nakao Yasuchika Takeishi Isao Kubota

AIMS High mobility group box 1 (HMGB1) is an abundant and ubiquitous nuclear DNA-binding protein that has multiple functions dependent on its cellular location. HMGB1 binds to DNA, facilitating numerous nuclear functions including maintenance of genome stability, transcription, and repair. However, little is known about the effects of nuclear HMGB1 on cardiac hypertrophy and heart failure. The ...

Journal: :Circulation research 2015
Christopher J Traynham Alessandro Cannavo Yan Zhou Alexandre G Vouga Benjamin P Woodall Jonathan Hullmann Jessica Ibetti Jessica I Gold J Kurt Chuprun Erhe Gao Walter J Koch

RATIONALE G protein-coupled receptor kinases (GRKs) are dynamic regulators of cellular signaling. GRK5 is highly expressed within myocardium and is upregulated in heart failure. Although GRK5 is a critical regulator of cardiac G protein-coupled receptor signaling, recent data has uncovered noncanonical activity of GRK5 within nuclei that plays a key role in pathological hypertrophy. Targeted ca...

2016
Yu‐yan Lu Da‐chun Xu Yi‐fan Zhao Guo‐fu Zhu Meng‐yun Zhu Wei‐jing Liu Xue‐jing Yu Wei Chen Zheng Liu Ya‐wei Xu

BACKGROUND Smad nuclear interacting protein 1 (SNIP1) plays a critical role in cell proliferation, transformation of embryonic fibroblasts, and immune regulation. However, the role of SNIP1 in cardiac hypertrophy remains unclear. METHODS AND RESULTS Here we examined the role of SNIP1 in pressure overload-induced cardiac hypertrophy and its mechanisms. Our results demonstrated that SNIP1 expre...

Journal: :Hypertension 2007
Hong-Liang Li Zhi-Gang She Tian-Bo Li Ai-Bing Wang Qinglin Yang Yu-Sheng Wei Yi-Guang Wang De-Pei Liu

Myofibrillogenesis regulator-1 (MR-1) augments cardiomyocytes hypertrophy induced by angiotensin II (Ang II) in vitro. However, its roles in cardiac hypertrophy in vivo remain unknown. Here, we investigate whether MR-1 can promote cardiac hypertrophy induced by Ang II in vivo and elucidate the molecular mechanisms of MR-1 on cardiac hypertrophy. We used a model of Ang II-induced cardiac hypertr...

Journal: :British heart journal 1985
M Been D Kean M A Smith R H Douglas J J Best A L Muir

The large differences in the spin lattice relaxation times (T1) of blood and myocardium (measured by nuclear magnetic resonance) allow the heart to be visualised without the use of contrast media. The findings using nuclear magnetic resonance in 11 unselected patients with hypertrophic cardiomyopathy were compared with those in equal numbers of normal subjects and patients with electrocardiogra...

Journal: :Hypertension 2014
Ding-Sheng Jiang Yu Liu Heng Zhou Yan Zhang Xiao-Dong Zhang Xiao-Fei Zhang Ke Chen Lu Gao Juan Peng Hui Gong Yingjie Chen Qinglin Yang Peter P Liu Guo-Chang Fan Yunzeng Zou Hongliang Li

Cardiac hypertrophy is a complex pathological process that involves multiple factors including inflammation and apoptosis. Interferon regulatory factor 7 (IRF7) is a multifunctional regulator that participates in immune regulation, cell differentiation, apoptosis, and oncogenesis. However, the role of IRF7 in cardiac hypertrophy remains unclear. We performed aortic banding in cardiac-specific I...

2017
Xiurong Li Yuhuai Lan Yan Wang Minghao Nie Yanhong Lu Eryang Zhao

Telmisartan, a type of angiotensin II (Ang II) receptor inhibitor, is a common agent used to treat hypertension in the clinic. Hypertension increases cardiac afterload and promotes cardiac hypertrophy. However, the ventricular Ang II receptor may be activated in the absence of hypertension. Therefore, telmisartan may reduce cardiac hypertrophy by indirectly ameliorating hypertensive symptoms an...

Journal: :The Journal of biological chemistry 2001
C Perez-Terzic A M Gacy R Bortolon P P Dzeja M Puceat M Jaconi F G Prendergast A Terzic

Each nuclear pore is responsible for both nuclear import and export with a finite capacity for bidirectional transport across the nuclear envelope. It remains poorly understood how the nuclear transport pathway responds to increased demands for nucleocytoplasmic communication. A case in point is cellular hypertrophy in which increased amounts of genetic material need to be transported from the ...

2015
Ersilia Cipolletta Maria Rosaria Rusciano Angela Serena Maione Gaetano Santulli Daniela Sorriento Carmine Del Giudice Michele Ciccarelli Antonietta Franco Catherine Crola Pietro Campiglia Marina Sala Isabel Gomez-Monterrey Nicola De Luca Bruno Trimarco Guido Iaccarino Maddalena Illario Xun Ai

AIMS Activation of Ca2+/Calmodulin protein kinase II (CaMKII) is an important step in signaling of cardiac hypertrophy. The molecular mechanisms by which CaMKII integrates with other pathways in the heart are incompletely understood. We hypothesize that CaMKII association with extracellular regulated kinase (ERK), promotes cardiac hypertrophy through ERK nuclear localization. METHODS AND RESU...

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