نتایج جستجو برای: neuroinflammatory

تعداد نتایج: 2211  

Journal: :مجله علوم اعصاب شفای خاتم 0
zeinab ghodsi pour department of clinical pharmacy, school of pharmacy, hamadan university of medical science, hamadan, iran fatemeh ghaedamini department of clinical pharmacy, school of pharmacy, hamadan university of medical science, hamadan, iran ghazal gholampour department of clinical pharmacy, school of pharmacy, hamadan university of medical science, hamadan, iran

more than 10 million people worldwide are living with parkinson’s disease (pd). the ages of people who have parkinson are variable, usually between 50 and 80 years, the average of them is 55 years old. the symptoms of pd are progressive, and within 10 to 20. it happens when dopaminergic neurons were being degenerate. peripheral and central inflammatory and oxidative stress pathways play a compl...

Journal: :Brain, behavior, and immunity 2016
Matthew G Frank Michael D Weber Laura K Fonken Sarah A Hershman Linda R Watkins Steven F Maier

The alarmin high mobility group box-1 (HMGB1) has been implicated as a key factor mediating neuroinflammatory processes. Recent findings suggest that the redox state of HMGB1 is a critical molecular feature of HMGB1 such that the reduced form (fr-HMGB1) is chemotactic, while the disulfide form (ds-HMGB1) is pro-inflammatory. The present study examined the neuroinflammatory effects of these mole...

2016
Matthew G. Frank Michael D. Weber Linda R. Watkins Steven F. Maier

Stress and glucocorticoids (GCs) have universally been considered to be anti-inflammatory, however in recent years, stress and GCs have been found to exert permissive effects (immunological priming) on neuroinflammatory processes. This phenomenon of priming is characterized by prior stress or GC exposure potentiating the neuroinflammatory response to a subsequent immune challenge. A considerabl...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2015
Scott J Webster Linda J Van Eldik D Martin Watterson Adam D Bachstetter

Epidemiological studies have associated increased risk of Alzheimer's disease (AD)-related clinical symptoms with a medical history of head injury. Currently, little is known about pathophysiology mechanisms linked to this association. Persistent neuroinflammation is one outcome observed in patients after a single head injury. Neuroinflammation is also present early in relevant brain regions du...

Ali Ebrahimi, Anahita Ghazaghi, Malihe Bahadoran, Mokhtar Ahmadi,

Depression alone is a severe and acute disease, but its close relationship with neuroinflammation and neurodegenerative diseases has emphasized its importance. In fact, the linking bridges between depression and these diseases are cytokines, especially pro-inflammatory cytokines which have fundamental roles in organizing different parts of central nervous system and emotions. Studying the relat...

Journal: :AJNR. American journal of neuroradiology 2008
R K Gupta K Nath A Prasad K N Prasad M Husain R K S Rathore N Husain C Srivastava P Khetan R Trivedi P A Narayana

BACKGROUND AND PURPOSE Neuroinflammatory molecules, including tumor necrosis factor-alpha, interleukin1-beta, lymphocyte function associated molecule-1, and intercellular cell adhesion molecule-1 contribute to the development of brain abscess. We hypothesized that the high fractional anisotropy (FA) in the brain abscess cavity reflects the upregulation of these neuroinflammatory molecules. MA...

Journal: :European Journal of Pain 2014

Journal: :Psychology and Behavioral Science International Journal 2017

Journal: :Neuroimmunology and Neuroinflammation 2015

Journal: :Cell reports 2015
Miguel Angel Burguillos Martina Svensson Tim Schulte Antonio Boza-Serrano Albert Garcia-Quintanilla Edel Kavanagh Martiniano Santiago Nikenza Viceconte Maria Jose Oliva-Martin Ahmed Mohamed Osman Emma Salomonsson Lahouari Amar Annette Persson Klas Blomgren Adnane Achour Elisabet Englund Hakon Leffler Jose Luis Venero Bertrand Joseph Tomas Deierborg

Inflammatory response induced by microglia plays a critical role in the demise of neuronal populations in neuroinflammatory diseases. Although the role of toll-like receptor 4 (TLR4) in microglia's inflammatory response is fully acknowledged, little is known about endogenous ligands that trigger TLR4 activation. Here, we report that galectin-3 (Gal3) released by microglia acts as an endogenous ...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید