نتایج جستجو برای: lrba deficiency

تعداد نتایج: 137277  

2017
Nina Bratanič Jernej Kovač Katka Pohar Katarina Trebušak Podkrajšek Alojz Ihan Tadej Battelino Magdalena Avbelj Stefanija

BACKGROUND Lipopolysaccharide-responsive, beige-like anchor protein (LRBA) deficiency is characterized by primary immunodeficiency and autoimmunity. Cancer may present another feature of LRBA deficiency. We describe a case history of a young adult with LRBA deficiency and two independent malignancies. METHODS Family-trio whole exome sequencing with unbiased phenotype ontology approach was use...

2017
Deborah L Burnett Ian A Parish Etienne Masle-Farquhar Robert Brink Christopher C Goodnow

Inherited mutations in lipopolysaccharide-responsive beige-like anchor (LRBA) cause a recessive human immune dysregulation syndrome with memory B-cell and antibody deficiency (common variable immunodeficiency), inflammatory bowel disease, enlarged spleen and lymph nodes, accumulation of activated T cells and multiple autoimmune diseases. To understand the pathogenesis of the syndrome, C57BL/6 m...

2017
Nashat Al Sukaiti Khwater AbdelRahman Jalila AlShekaili Sumaya Al Oraimi Aisha Al Sinani Nasser Al Rahbi Vicky Cho Matt Field Matthew C Cook

Mutations in lipopolysaccharide-responsive vesicle trafficking, beach and anchor-containing protein (LRBA) cause immune deficiency and inflammation. Here, we are reporting a novel homozygous mutation in LRBA allele in 7-year-old Omani boy, born to consanguineous parents. He presented with type 1 diabetes, autoimmune haematological cytopenia, recurrent chest infections and lymphocytic interstiti...

Journal: :American journal of human genetics 2012
Gabriela Lopez-Herrera Giacomo Tampella Qiang Pan-Hammarström Peer Herholz Claudia M Trujillo-Vargas Kanchan Phadwal Anna Katharina Simon Michel Moutschen Amos Etzioni Adi Mory Izhak Srugo Doron Melamed Kjell Hultenby Chonghai Liu Manuela Baronio Massimiliano Vitali Pierre Philippet Vinciane Dideberg Asghar Aghamohammadi Nima Rezaei Victoria Enright Likun Du Ulrich Salzer Hermann Eibel Dietmar Pfeifer Hendrik Veelken Hans Stauss Vassilios Lougaris Alessandro Plebani E Michael Gertz Alejandro A Schäffer Lennart Hammarström Bodo Grimbacher

Most autosomal genetic causes of childhood-onset hypogammaglobulinemia are currently not well understood. Most affected individuals are simplex cases, but both autosomal-dominant and autosomal-recessive inheritance have been described. We performed genetic linkage analysis in consanguineous families affected by hypogammaglobulinemia. Four consanguineous families with childhood-onset humoral imm...

Journal: :Acta medica Iranica 2016
Sima Shokri Mohammad Nabavi Tatjana Hirschmugl Asghar Aghamohammadi Saba Arshi Mohamad Hassan Bemanian Morteza Fallahpour Rasool Molatefi Mahsa Rekabi Narges Eslami Javad Ahmadian Kian Darabi Gholam Reza Sedighi Maryam Monajemzadeh Mohammadreza Modaresi Nima Parvaneh Kaan Boztug Nima Rezaei

LPS-Responsive Beige-like Anchor (LRBA) deficiency is a disease which has recently been described in a group of patients with common variable immunodeficiency (CVID) in association with autoimmunity and/or inflammatory bowel disease (IBD)-like phenotype. We here describe a 10-year-old boy who experienced recurrent infections, mainly in the respiratory system, associated with thrombocytopenia an...

2016
Shahrzad Bakhtiar Frank Ruemmele Fabienne Charbit-Henrion Eva Lévy Frédéric Rieux-Laucat Nadine Cerf-Bensussan Peter Bader Ulrich Paetow

Monogenic primary immunodeficiency syndromes can affect one or more endocrine organs by autoimmunity during childhood. Clinical manifestations include type 1 diabetes mellitus, hypothyroidism, adrenal insufficiency, and vitiligo. Lipopolysaccharide (LPS)-responsive beige-like anchor protein (LRBA) deficiency was described in 2012 as a novel primary immunodeficiency, predominantly causing immune...

Journal: :acta medica iranica 0
sima shokri department of allergy and clinical immunology, rasool-e-akram hospital, iran university of medical sciences, tehran, iran. mohammad nabavi department of allergy and clinical immunology, rasool-e-akram hospital, iran university of medical sciences, tehran, iran. tatjana hirschmugl cemm research center for molecular medicine, austrian academy of sciences, vienna, austria. asghar aghamohammadi research center for immunodeficiencies, children's medical center, tehran university of medical sciences, tehran, iran. saba arshi department of allergy and clinical immunology, rasool-e-akram hospital, iran university of medical sciences, tehran, iran. mohamad hassan bemanian department of allergy and clinical immunology, rasool-e-akram hospital, iran university of medical sciences, tehran, iran.

lps-responsive beige-like anchor (lrba) deficiency is a disease which has recently been described in a group of patients with common variable immunodeficiency (cvid) in association with autoimmunity and/or inflammatory bowel disease (ibd)-like phenotype. we here describe a 10-year-old boy who experienced recurrent infections, mainly in the respiratory system, associated with thrombocytopenia an...

2017
Shahrzad Bakhtiar Laura Gámez-Díaz Andrea Jarisch Jan Soerensen Bodo Grimbacher Bernd Belohradsky Klaus-Michael Keller Christoph Rietschel Thomas Klingebiel Sibylle Koletzko Michael H. Albert Peter Bader

Inflammatory bowel disease (IBD) in young children can be a clinical manifestation of various primary immunodeficiency syndromes. Poor clinical outcome is associated with poor quality of life and high morbidity from the complications of prolonged immunosuppressive treatment and malabsorption. In 2012, mutations in the lipopolysaccharide-responsive beige-like anchor (LRBA) gene were identified a...

Journal: :Russian Journal of Allergy 2020

Journal: :Frontiers in Immunology 2015

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