نتایج جستجو برای: janus kinase 2

تعداد نتایج: 2686970  

ژورنال: :کومش 0
خدیجه کوشکی khadijeh koushki basic and molecular epidemiology of gastrointestinal disorders research center, research institute for gastroenterology and liver diseases,shahidbeheshti university of medical sciences, tehran, iranمرکز تحقیقات بیماری های گوارش و کبد، پژوهشکده تحقیقات بیماری های گوارش و کبد، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران محمد رستمی نژاد mohammad rostami nejad basic and molecular epidemiology of gastrointestinal disorders research center, research institute for gastroenterology and liver diseases,shahidbeheshti university of medical sciences, tehran, iranمرکز تحقیقات بیماری های گوارش و کبد، پژوهشکده تحقیقات بیماری های گوارش و کبد، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران پدرام عظیم زاده pedrami azimzadeh gastroenterology and liver diseases research center, research institute for gastroenterology and liver diseases,shahidbeheshti university of medical sciences, tehran, iranمرکز تحقیقات علوم پایه و مولکولار اپیدمیولوژی دستگاه گوارش، پژوهشکده تحقیقات بیماری های گوارش و کبد، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران حمید اسدزاده عقدایی hamid asadzadeh aghdaei gastroenterology and liver diseases research center, research institute for gastroenterology and liver diseases,shahidbeheshti university of medical sciences, tehran, iranمرکز تحقیقات علوم پایه و مولکولار اپیدمیولوژی دستگاه گوارش، پژوهشکده تحقیقات بیماری های گوارش و کبد، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران عباس حاج فتحعلی abas hajfathali taleghani hospital, shahidbeheshti university of medical sciences, tehran, iranبیمارستان آیت الله طالقانی، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران محمد امین پور حسینقلی mohammad amin pourhosseingholi basic and molecular epidemiology of gastrointestinal disorders research center, research institute for gastroenterology and liver diseases,shahidbeheshti university of medical sciences, tehran, iranمرکز تحقیقات بیماری های گوارش و کبد، پژوهشکده تحقیقات بیماری های گوارش و کبد، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران هدیه بالایی

سابقه و هدف: بیماری التهابی روده (inflammatory bowel disease)، شامل کولیت اولسراتیو (ulcerative colitis) و بیماری کرون (crohn’s disease)، یک اختلال التهابی مزمن دستگاه گوارش است که مطالعات گسترده ژنومی (genome-wide association study) نشان می دهد که برخی از ژن های مسیر اینترلوکین-23/t کمکی 17 (il-23/th17) مانند جانوس کیناز -2 (jak2) مستعدکننده ابتلا به بیماری التهابی روده هستند. هاپلوتایپ 46/1 ی...

Journal: :Bioorganic & medicinal chemistry letters 2014
Mark E McDonnell Haiyan Bian Jay Wrobel Garry R Smith Shuguang Liang Haiching Ma Allen B Reitz

We designed a series of anilino-indoylmaleimides based on structural elements from literature JAK3 inhibitors 3 and 4, and our lead 5. These new compounds were tested as inhibitors of JAKs 1, 2 and 3 and TYK2 for therapeutic intervention in rheumatoid arthritis (RA). Our requirements, based on current scientific rationale for optimum efficacy against RA with reduced side effects, was for potent...

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2010

Journal: :Cancer discovery 2012
Rita Andraos Zhiyan Qian Débora Bonenfant Joëlle Rubert Eric Vangrevelinghe Clemens Scheufler Fanny Marque Catherine H Régnier Alain De Pover Hugues Ryckelynck Neha Bhagwat Priya Koppikar Aviva Goel Lorenza Wyder Gisele Tavares Fabienne Baffert Carole Pissot-Soldermann Paul W Manley Christoph Gaul Hans Voshol Ross L Levine William R Sellers Francesco Hofmann Thomas Radimerski

Janus kinase (JAK) inhibitors are being developed for the treatment of rheumatoid arthritis, psoriasis, myeloproliferative neoplasms, and leukemias. Most of these drugs target the ATP-binding pocket and stabilize the active conformation of the JAK kinases. This type I binding mode can lead to an increase in JAK activation loop phosphorylation, despite blockade of kinase function. Here we report...

2013
Karoline Gäbler Catherine Rolvering Jakub Kaczor René Eulenfeld Sergio Álvarez Méndez Guy Berchem Valérie Palissot Iris Behrmann Claude Haan

The Janus kinase 2 mutant V617F occurs with high frequency in myeloproliferative neoplasms. Further mutations affecting the Janus kinase family have been discovered mostly in leukaemias and in myeloproliferative neoplasms. Owing to their involvement in neoplasia, inflammatory diseases and in the immune response, Janus kinases are promising targets for kinase inhibitor therapy in these disease s...

2017
L.C.S. De Vries M.E. Wildenberg W.J. De Jonge G.R. D’Haens

Inflammatory bowel diseases, such as ulcerative colitis and Crohn's disease, are disabling conditions characterised by chronic, relapsing inflammation of the gastrointestinal tract. Current treatments are not universally effective or, in the case of therapeutic antibodies, are hampered by immune responses. Janus kinase inhibitors are orally delivered small molecules that target cytokine signall...

Journal: :Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis 2008
Mario Lapecorella Antonio Tabilio Alessandro Lucchesi Mariasanta Napolitano Marianna Colagrande Mauro Di Ianni Guglielmo Mariani

The discovery of the Janus kinase 2 Val617Phe mutation has brought new insights into the development of myeloproliferative disorders; however, the pathogenesis of essential thrombocythemia and its related thrombotic complications has not been completely understood. Although the Janus kinase 2 Val617Phe mutation confirms the initially suspected clonal character of the disease, factors influencin...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1998
R J Duhé G A Evans R A Erwin R A Kirken G W Cox W L Farrar

The activation of Janus kinases (JAKs) is crucial for propagation of the proliferative response initiated by many cytokines. The proliferation of various cell lines, particularly those of hematopoietic origin, is also modulated by mediators of oxidative stress such as nitric oxide and thiol redox reagents. Herein we demonstrate that nitric oxide and other thiol oxidants can inhibit the autokina...

Journal: :Haematologica 2014
Hajnalka Andrikovics Tunde Krahling Katalin Balassa Gabriella Halm Andras Bors Magdalena Koszarska Arpad Batai Janos Dolgos Judit Csomor Miklos Egyed Andrea Sipos Peter Remenyi Attila Tordai Tamas Masszi

Somatic insertions/deletions in the calreticulin gene have recently been discovered to be causative alterations in myeloproliferative neoplasms. A combination of qualitative and quantitative allele-specific polymerase chain reaction, fragment-sizing, high resolution melting and Sanger-sequencing was applied for the detection of three driver mutations (in Janus kinase 2, calreticulin and myelopr...

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