نتایج جستجو برای: glioblastomamultiforme

تعداد نتایج: 18  

Journal: :Annals of Pathology and Laboratory Medicine 2017

2015
Shashikant Adlekha Tandra Chadha A. Ragunath B. Sumangala Robin George

Glioblastoma, the most frequent primary brain tumor and the most malignant neoplasm, can have the most heterogeneous morphological presentation. Along with classical morphological features, areas of oligodendroglial component and mesenchymal differentiation may be seen. The present case was a rare case of glioblastoma presenting with sarcomatous and oligodendroglial components. The case was dia...

Journal: :iranian journal of radiation research 0
h. acar selcuk university, faculty of medicine, department of radiation oncology, konya, turkey g. yavas selcuk university, faculty of medicine, department of radiation oncology, konya, turkey c. yavas konya training and research hospital, department of radiation oncology, konya, turkey

background: broca’s and wernicke’s areas, which are important language areas of brain, are commonly irradiated in patients with right frontal lobe glioblastomamultiforme (gbm). we aimed to compare two different treatment planning techniques in terms of the doses in the broca’s and wernicke’s areas for the patients with right frontal gbm. materials and methods: two different treatment planning t...

2013
J. Zachary Sanborn Sofie R. Salama Mia Grifford Cameron W. Brennan Tom Mikkelsen Suresh Jhanwar Sol Katzman Lynda Chin David Haussler

DNA sequencing offers a powerful tool in oncology based on the precise definition of structural rearrangements and copy number in tumor genomes. Here, we describe the development of methods to compute copy number and detect structural variants to locally reconstruct highly rearranged regions of the tumor genome with high precision from standard, short-read, paired-end sequencing datasets.Wefind...

2012
Yi Chieh Lim Tara L. Roberts Bryan W. Day Angus Harding Sergei Kozlov Amanda W. Kijas Kathleen S. Ensbey David G. Walker Martin F. Lavin

Glioblastomamultiforme (GBM) is themost common formofbrain tumorwith apoorprognosis and resistance toradiotherapy.Recentevidencesuggests thatglioma-initiatingcellsplayacentral role inradioresistance through DNAdamage checkpoint activation and enhanced DNA repair. To investigate this inmore detail, we compared the DNA damage response in nontumor forming neural progenitor cells (NPC) and glioma-i...

2012
Shinji Kohsaka Lei Wang Kazuhiro Yachi Roshan Mahabir Takuhito Narita Tamio Itoh Mishie Tanino Taichi Kimura Hiroshi Nishihara Shinya Tanaka

Glioblastomamultiforme (GBM) is one of themost aggressive human tumorswith apoor prognosis. Current standard treatment includes chemotherapy with the DNA-alkylating agent temozolomide concomitant with surgical resection and/or irradiation.However, anumber of cases are resistant to temozolomide-inducedDNA damage due to elevated expression of the DNA repair enzyme O-methylguanine-DNA methyltransf...

2009
A.K.M. Ghulam Muhammad Marianela Candolfi Gwendalyn D. King Kader Yagiz David Foulad Yohei Mineharu Kurt M. Kroeger Katherine A. Treuer W. Stephen Nichols Nicholas S. Sanderson Jieping Yang Maksim Khayznikov Nico Van Rooijen Pedro R. Lowenstein Maria G. Castro

Purpose: Glioblastoma multiforme is a deadly primary brain cancer. Because the tumor kills due to recurrences, we tested the hypothesis that a new treatment would lead to immunological memory in a rat model of recurrent glioblastoma multiforme. Experimental Design: Wedeveloped a combined treatment using an adenovirus (Ad) expressing fms-like tyrosine kinase-3 ligand (Flt3L), which induces the i...

2011
Linda C. Hsi Suman Kundu Juan Palomo Bo Xu Ryan Ficco Michael A. Vogelbaum Martha K. Cathcart

Glioblastomamultiforme (GBM) is one of the most lethal forms of cancer, with a survival rate of only 13% to 27%within 2 years of diagnosis despite optimal medical treatment. We hypothesize that the presence of a unique IL-13Ra2 decoy receptor prevents GBM apoptosis. This receptor has a high affinity for interleukin13 (IL-13), binds the cytokine, and competitively inhibits the intracellular sign...

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