نتایج جستجو برای: facioscapulohumeralmuscular dystrophy

تعداد نتایج: 22674  

Journal: :iranian journal of neurology 0
marjan asadollahi loghman hospital, department of neurology, shahid beheshti university of medical sciences, tehran, iran. bibiseyedeh rezaiyan loghman hospital, department of neurology, shahid beheshti university of medical sciences, tehran, iran. hiva amjadi loghman hospital, department of neurology, shahid beheshti university of medical sciences, tehran, iran.

facioscapulohumeral muscular dystrophy (fshd) is a common inherited muscular dystrophy presented clinically with slowly progressive weakness and wasting of facial and limb muscles and rare bulbar muscle involvement. we present herein a 70-year-old man who was a known case of fshd with complaint of 15-day history of progressive difficulty in chewing and dysarthria and was found to have myastheni...

2015
Jeffrey M. Statland Karen J. Odrzywolski Bharati Shah Don Henderson Alex F. Fricke Silvére M. van der Maarel Stephen J. Tapscott Rabi Tawil

BACKGROUND Posited pathological mechanisms in Facioscapulohumeral Muscular Dystrophy (FSHD) include activation in somatic tissue of normally silenced genes, increased susceptibility to oxidative stress, and induction of apoptosis. OBJECTIVE To determine the histopathological changes in FSHD muscle biopsies and compare to possible pathological mechanisms of disease. METHODS We performed a cr...

Journal: :Human mutation 2015
Francesca Puppo Eugenie Dionnet Marie-Cécile Gaillard Pascaline Gaildrat Christel Castro Catherine Vovan Karine Bertaux Rafaelle Bernard Shahram Attarian Kanako Goto Ichizo Nishino Yukiko Hayashi Frédérique Magdinier Martin Krahn Françoise Helmbacher Marc Bartoli Nicolas Lévy

Facioscapulohumeralmuscular dystrophy (FSHD) is linked to copy-number reduction (N < 10) of the 4q D4Z4 subtelomeric array, in association with DUX4-permissive haplotypes. This main form is indicated as FSHD1. FSHD-like phenotypes may also appear in the absence of D4Z4 copy-number reduction. Variants of the SMCHD1 gene have been reported to associate with D4Z4 hypomethylation in DUX4-compatible...

ژورنال: بیهوشی و درد 2013

Aim and Background: Becker’s muscular dystrophy (BMD) is similar to the Duchenne’s muscular dystrophy, but the clinical course is milder.We introduced a patient with Becker Muscle Dystrophy who candidate for emergency cesarean section.Case report:A36-year-old woman, gestational age 31 weeks, with a history of Becker Muscle Dystrophy and fetal distress who candidate for emergency cesarean sectio...

Journal: :iranian journal of child neurology 0
mohammad barzegar 1. pediatric health research center, tabriz university of medical sciences, tabriz, iran. 2. division of pediatric neurology, tabriz children’s hospital, tabriz university of medical sciences, tabriz, iran parinaz habibi 1. pediatric health research center, tabriz university of medical sciences, tabriz, iran. mortaza mortaza bonyady 3. center of excellence for biodiversity, department of molecular medical genetics, faculty of natural sciences university of tabriz, tabriz, iran vahideh topchizadeh 4. physical medicine &amp; rehabilitation research center, tabriz university of medical sciences, tabriz, iran shadi shiva* 1. pediatric health research center, tabriz university of medical sciences, tabriz, iran. 2. division of pediatric neurology, tabriz children’s hospital, tabriz university of medical sciences, tabriz, iran

how to cite this article: barzegar m, habibi p, bonyady m, topchizadeh v, shiva sh. exon deletion pattern in duchene muscular dystrophy in north west of iran. iran j child neurol. 2015 winter; 9(1): 42-48. abstract objective duchene and becker muscular dystrophy (dmd/ bmd) are x-linked disorders that both are the result of heterogeneous mutations in the dystrophin gene. the frequency and distri...

Journal: :genetics in the 3rd millennium 0
غلامرضا زمانی gholamreza zamani child neurologist, children&apos;s medical center, tehran university of medical sciences (tums)

muscular dystrophies are inherited disorders that cause progressive muscle weakness (myopathy) and atrophy (loss of muscle mass) due to defects in one or more genes required for normal muscle function. some of the genes responsible for these conditions have been identified .there are a number of different types of muscular dystrophy. the primary symptom for most types is muscle weakness, althou...

Journal: :acta medica iranica 0
fardeen ali malayeri department of neurogenetics, iranian center of neurological research, imam khomeini hospital , tehran university of medical sciences, tehran, iran. and department of clinical biochemistry, isfahan pharmaceutical sciences research center, school of pharmacy and pharmaceutical sciences, isfahan university of medical sciences, isfahan, iran. mojtaba panjehpour department of clinical biochemistry, isfahan pharmaceutical sciences research center, school of pharmacy and pharmaceutical sciences, isfahan university of medical sciences, isfahan, iran. ahmad movahedian department of clinical biochemistry, isfahan pharmaceutical sciences research center, school of pharmacy and pharmaceutical sciences, isfahan university of medical sciences, isfahan, iran. majid ghaffarpour iranian center of neurological research, imam khomeini hospital, tehran university of medical sciences, tehran, iran. gholam reza zamani department of neurology, children medical center, school of medicine, tehran university of medical sciences, tehran, iran. hajifaraj tabrizi department of medical genetics, school of medicine, tehran university of medical sciences, tehran, iran.

this study determines the value of linkage analysis using six rflp markers for carrier detection and prenatal diagnosis in familial dmd/bmd cases and their family members for the first time in the iranian population. we studied the dystrophin gene in 33 unrelated patients with clinical diagnosis of dmd or bmd. subsequently, we determined the rate of heterozygosity for six intragenic rflp marker...

Dabiri Shahriar Meymandi Simin Shamsi Shafiei Hamidreza

Epidermolysis Bullosa Puriginosa is a genetic mechanobullous disease characterized by pruritus, lichenified or nodular prurigo-like lesions, occasional trauma-induced blistering, excoriations, milia, nail dystrophy and albopapuloid lesions that appear at birth or later. Scarring and prurigo are most prominent on the shins. Herein, we report a case with a history of blisters since childhood foll...

Fahimeh Abdollahimajd, Kani Zahra Asadi Reza Jaffari Fesharaki Seyed-Mostafa Razavi Shahidi-Dadras Mohammad Yousefi Maryam

We hereby report a 79-year-old Iranian man presenting with nail dystrophy and subsequent development of purpuric and ecchymotic plaques, hemorrhagic bullae, and infiltrated papules on the head, neck and trunk. Histological examination of the gingiva, bone marrow aspiration, and biopsy confirmed the diagnosis of primary systemic amyloidosis. In this case, nail dystrophy was the presenting sign o...

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