abstract beet soilborne virus (bsbv) is a member of the genus pomovirus, with rod shaped particles and three plus single - stranded of rna. bsbv is morphologically similar to beet necrotic yellow vein virus (bnyvv) and both are transmitted by the soilborne fungus, polymyxa betae keskin that survives in soil for many years. host range of bsbv is limited to the family of chenopodiaceae. in order ...

Three soilborne viruses transmitted by Polymyxa betae KESKIN in sugar beet have been described: Beet necrotic yellow vein virus (BNYVV), the agent of rhizomania, Beet soilborne virus (BSBV), and Beet virus Q (BVQ). A multiplex reverse transcription-PCR technique was developed to simultaneously detect BNYVV, BSBV, and BVQ, together with their vector, P. betae. The detection threshold of the test...

Sugar Beet Nematode (SBN), Heterodera schachtii, is one of the most destructive pests of sugar beets worldwide. It is present in all sugar beet growing areas of Wyoming and is particularly severe in fields located in close proximity to processing plants where sugar beets have been grown the longest. This soilborne, parasitic nematode attacks and destroys the small feeder roots of sugar beet pla...

Evaluation of Rhizoctonia zeae as a potential biological control option for fungal root diseases of sugar beet" (2015). Abstract Several common root diseases routinely damage sugar beet in Nebraska and other production areas of the Central High Plains, and it is becoming more common to find fields infested simultaneously with multiple pathogens. Owing to the shortage of available fungicides for...

Beet necrotic yellow vein virus (BNYVV) and Beet soilborne mosaic virus (BSBMV) are members of the genus Benyvirus (9). Both viruses infect members of the family Chenopodiaceae, most notably sugar beet (Beta vulgaris L.), and are transmitted by the plasmodiophorid, Polymyxa betae (1,2). BNYVV infection causes rhizomania disease in sugar beet (4,21); however, BSBMV infection does not (28). BSBMV...

Invasive soilborne plant pathogens cause substantial damage to crops and natural populations, but our understanding of how to prevent their epidemics or reduce their damage is limited. A key and experimentally-tested concept in the epidemiology of soilborne plant diseases is that of a threshold spacing between hosts below which epidemics (invasive spread) can occur. We extend this paradigm by e...