نتایج جستجو برای: glutamic acid decarboxylase autoantibodies gad65

تعداد نتایج: 768647  

2017
Anders Persson Charlotte Becker Ida Hansson Anita Nilsson Carina Törn

Citation for published version (APA): Persson, A., Becker, C., Jönsson, I., Ramelius, A., & Törn, C. (2010). Comparison of measurements of autoantibodies to glutamic acid decarboxylase and islet antigen-2 in whole blood eluates from dried blood spots using the RSR-enzyme linked immunosorbent assay kits and in-house radioimmunoassays. Experimental Diabetes Research, 2010, [173652]. DOI: 10.1155/...

Journal: :Frontiers in cellular neuroscience 2016
Jana K. Hackert Lorenz Müller Marco Rohde Christian G. Bien Rüdiger Köhling Timo Kirschstein

Glutamic acid decarboxylase of 65 kDa (GAD65) antibodies have been reported in a variety of neurological disorders such as stiff-person syndrome (SPS), sporadic ataxia and some cases of epilepsy. Since the target is believed to be the cytoplasmic enzyme GAD65, the key enzyme of γ-aminobutyric acid (GABA) synthesis, the pathophysiological role of these antibodies is poorly understood. Here, we s...

ژورنال: یافته 2017

Background: Latent autoimmune diabetes of adults (LADA) is a slowly progressive form of diabetes managed with diet and oral hypoglycemic agents before becoming insulin requiring. Islet autoantibodies are used for differential diagnosis between LADA and Type 2 diabetes mellitus. The aims of this study were to investigate the frequency of LADA using islet autontibodies in population with T2DM and...

2018
Kenneth N. Fish Brad R. Rocco David A. Lewis

In human prefrontal cortex (PFC), ~85% of γ-aminobutyric acid (GABA)-expressing neurons can be subdivided into non-overlapping groups by the presence of calbindin (CB), calretinin (CR) or parvalbumin (PV). Substantial research has focused on the differences in the laminar locations of the cells bodies of these neurons, with limited attention to the distribution of their axon terminals, their si...

Journal: :The Journal of Experimental Medicine 2002
Kristin V. Tarbell Mark Lee Erik Ranheim Cheng Chi Chao Maija Sanna Seon-Kyeong Kim Peter Dickie Luc Teyton Mark Davis Hugh McDevitt

Glutamic acid decarboxylase (GAD)65 is an early and important antigen in both human diabetes mellitus and the nonobese diabetic (NOD) mouse. However, the exact role of GAD65-specific T cells in diabetes pathogenesis is unclear. T cell responses to GAD65 occur early in diabetes pathogenesis, yet only one GAD65-specific T cell clone of many identified can transfer diabetes. We have generated tran...

Journal: :Developmental neurobiology 2015
Csaba Vastagh Marija Schwirtlich Andrea Kwakowsky Ferenc Erdélyi Frank L Margolis Yuchio Yanagawa Zoya Katarova Gábor Szabó

Gamma-aminobutyric acid (GABA) has a dual role as an inhibitory neurotransmitter in the adult central nervous system (CNS) and as a signaling molecule exerting largely excitatory actions during development. The rate-limiting step of GABA synthesis is catalyzed by two glutamic acid decarboxylase isoforms GAD65 and GAD67 coexpressed in the GABAergic neurons of the CNS. Here we report that the two...

Journal: :Annals of the New York Academy of Sciences 2005
Caroline Tilikete Alain Vighetto Paul Trouillas Jérome Honnorat

Glutamic acid decarboxylase (GAD) catalyzes the conversion of glutamic acid to gamma-aminobutyric acid (GABA). Autoantibodies directed against GAD (antiGAD-Ab) have been described in patients with insulin-dependent diabetes mellitus, stiff-man syndrome, and in a few patients with progressive cerebellar ataxia. The presence of these autoantibodies suggests an autoimmune pathophysiological mechan...

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