نتایج جستجو برای: chloride channel

تعداد نتایج: 305756  

Journal: :European Journal of Cell Biology 2000

Journal: :General physiology and biophysics 1991
P Proks O Hurnák J Zachar

Low-conductance chloride channel from skeletal muscle SR vesicles of the crayfish Astacus fluviatilis was incorporated into planar lipid bilayers and its basic characteristics were investigated. The channel has a relatively low unitary conductance of 26 pS in symmetrical 160 mmol/l choline-chloride. The dependence of the channel conductance on Cl- concentration shows saturating behavior with a ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1996
P Hart J D Warth P C Levesque M L Collier Y Geary B Horowitz J R Hume

cAMP-dependent chloride channels in heart contribute to autonomic regulation of action potential duration and membrane potential and have been inferred to be due to cardiac expression of the epithelial cystic fibrosis transmembrane conductance regulator (CFTR) chloride channel. In this report, a cDNA from rabbit ventricle was isolated and sequenced, which encodes an exon 5 splice variant (exon ...

2015
Zenon Trela Zbigniew Burdach Agnieszka Siemieniuk Stanisław Przestalski Waldemar Karcz Zhong-Hua Chen

In the present study, patch-clamp techniques have been used to investigate the effect of trimethyltin chloride (Met3SnCl) on the slow vacuolar (SV) channels in vacuoles from red beet (Beta vulgaris L.) taproots. Activity of SV channels has been measured in whole-vacuole and cytosolic side-out patch configurations. It was found that addition of trimethyltin chloride to the bath solution suppress...

Journal: :The Journal of clinical investigation 2016
Axel R Concepcion Martin Vaeth Larry E Wagner Miriam Eckstein Lee Hecht Jun Yang David Crottes Maximilian Seidl Hyosup P Shin Carl Weidinger Scott Cameron Stuart E Turvey Thomas Issekutz Isabelle Meyts Rodrigo S Lacruz Mario Cuk David I Yule Stefan Feske

Eccrine sweat glands are essential for sweating and thermoregulation in humans. Loss-of-function mutations in the Ca2+ release-activated Ca2+ (CRAC) channel genes ORAI1 and STIM1 abolish store-operated Ca2+ entry (SOCE), and patients with these CRAC channel mutations suffer from anhidrosis and hyperthermia at high ambient temperatures. Here we have shown that CRAC channel-deficient patients and...

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