نتایج جستجو برای: 10 jun

تعداد نتایج: 1030171  

Journal: :Annals of Hepato-Biliary-Pancreatic surgery 2023

Ye Won JEON, Woohyung LEE*, Yejong PARK, Bong Jun KWAK, Eunsung JUN, Ki Byung SONG, Dae Wook HWANG, Jae Hoon LEE, Song Cheol KIM. Ann Hepatobiliary Pancreat Surg 2023;27:S62. https://doi.org/10.14701/ahbps.2023S1.OP-1-4

Journal: :American journal of physiology. Heart and circulatory physiology 2000
M Sato G A Cordis N Maulik D K Das

The role of stress-activated protein kinases (SAPKs), c-Jun NH(2)-terminal kinase (JNK) and p38 mitogen-activated protein (MAP) kinase, in preconditioning (PC) was examined with the use of isolated rat hearts subjected to four cyclic episodes of 5-min ischemia and 10-min reperfusion followed by 30-min ischemia and 2-h reperfusion (I/R). A group of hearts was preperfused with 100 microM curcumin...

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2011

Journal: :Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1997
M Nilsson J Ford S Bohm R Toftgård

We have previously identified a 20-bp sequence that mediates induced transcription in response to EGF, Ras, and Raf but not after TPA or UV stimulation. This composite response element, present in a long terminal repeat of a member within the VL30 family of retrotransposons, contains an AP-1-like site that cooperates in function with a juxtaposed sequence unrelated to known transcription factor...

Journal: :American journal of respiratory cell and molecular biology 2008
Renli Qiao Weihong Yan Carlos Clavijo Ruty Mehrian-Shai Qian Zhong Kwang-Jin Kim David Ann Edward D Crandall Zea Borok

Rat alveolar epithelial cells (AEC) in primary culture transdifferentiate from a type II (AT2) toward a type I (AT1) cell-like phenotype, a process that can be both prevented and reversed by keratinocyte growth factor (KGF). Microarray analysis revealed that these effects of KGF are associated with up-regulation of key molecules in the mitogen-activated protein kinase (MAPK) pathway. To further...

Journal: :Cancer research 2007
Krishna Murthi Vasudevan Ravshan Burikhanov Anindya Goswami Vivek M Rangnekar

Ras is one of the most commonly mutated oncogenes in the array of human cancers. The mechanism by which Ras induces cellular transformation is, however, not fully elucidated. We present here evidence that oncogenic Ras suppresses the expression of the tumor suppressor phosphatase and tensin homologue deleted from chromosome 10 (PTEN), and this action of oncogenic Ras is mediated by the Raf-mito...

Journal: :The Journal of biological chemistry 1995
H Sawai T Okazaki H Yamamoto H Okano Y Takeda M Tashima H Sawada M Okuma H Ishikura H Umehara

Ceramide has emerged as a novel lipid mediator in cell proliferation, differentiation, and apoptosis. In this work, we demonstrate that the levels of c-jun mRNA, c-Jun protein, and DNA binding activity of a nuclear transcription factor AP-1 to 12-o-tetradecanoylphorbol 13-acetate responsive elements all increased following treatment with the cell-permeable ceramide, N-acetylsphingosine in human...

Journal: :Molecular human reproduction 1996
A Salmi M Ammälä E M Rutanen

Oestrogen is the major stimulatory factor in endometrial cell proliferation. Animal and in-vitro studies have shown that proto-oncogenes c-fos and c-jun are regulated by oestrogen receptor (ER) complex. We have previously shown by Northern blot analysis that proto-oncogenes c-fos and c-jun are strongly expressed in human proliferative and early to mid-secretory endometrium. In this study, we ex...

2012
Saima E. Ferraris Kimmo Isoniemi Elin Torvaldson Julius Anckar Jukka Westermarck John E. Eriksson

The AP-1 transcription factor c-Jun has been shown to be essential for stress-induced apoptosis in several models. However, the molecular mechanisms underlying the proapoptotic activity of c-Jun are poorly understood. We identify the apoptosis-antagonizing transcription factor (AATF) as a novel nucleolar stress sensor, which is required as a cofactor for c-Jun-mediated apoptosis. Overexpression...

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