نتایج جستجو برای: kruppel

تعداد نتایج: 729  

Journal: :Cancer research 2000
K Kuramoto T Uesaka A Kimura M Kobayashi H Watanabe O Katoh

Vascular endothelial growth factor (VEGF) inhibits radiation-induced apoptosis in the leukemia cell line, CMK86 (O. Katoh et al., Cancer Res., 55: 5687-5692, 1995). To elucidate the molecular mechanisms underlying this inhibitory effect of VEGF, we attempted to identify a transcription factor involved in the cellular response to VEGF stimulation. We cloned the cDNA of a novel Kruppel-type zinc ...

Journal: :Current Biology 2016
Ezzat El-Sherif Michael Levine

Drosophila patterning genes often contain pairs of primary and shadow enhancers that possess overlapping activities [1-5]. It has been suggested that this regulatory "redundancy" helps ensure reliable activation of gene expression under stressful conditions such as increases in temperature [4, 5]. There is also evidence that shadow enhancers help produce sharp on/off boundaries of gene expressi...

Journal: :Proceedings of the National Academy of Sciences 2007

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2014
Jun-ichi Abe Bradford C Berk

Atherosclerosis is a focal disease that develops preferentially where nonlaminar, disturbed blood flow occurs, such as branches, bifurcations, and curvatures of large arteries. Endothelial cells sense and respond differently to disturbed flow compared with steady laminar flow. Disturbed flow that occurs in so-called atheroprone areas activates proinflammatory and apoptotic signaling, and this r...

Journal: :The Journal of clinical investigation 2013
Yuan Lu Lisheng Zhang Xudong Liao Panjamaporn Sangwung Domenick A Prosdocimo Guangjin Zhou Alexander R Votruba Leigh Brian Yuh Jung Han Huiyun Gao Yunmei Wang Koichi Shimizu Kaitlyn Weinert-Stein Maria Khrestian Daniel I Simon Neil J Freedman Mukesh K Jain

Activation of cells intrinsic to the vessel wall is central to the initiation and progression of vascular inflammation. As the dominant cellular constituent of the vessel wall, vascular smooth muscle cells (VSMCs) and their functions are critical determinants of vascular disease. While factors that regulate VSMC proliferation and migration have been identified, the endogenous regulators of VSMC...

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