نتایج جستجو برای: chronic allograft dysfunction

تعداد نتایج: 644743  

2007
Yong Loo Lin Yukio Ando

Most problematic liver allograft biopsies are obtained more than year after transplantation, due largely to overlapping clinical, serological and histopathological features of native disease recurrence and other potential causes of late allograft dysfunction. More than one insult can be responsible. About 75% of biopsies from long-surviving recipients with abnormal liver function tests/symptoms...

Introduction:Detecting renal allograft dysfunction early will allow timely diagnosis and treatment. There is no objective recommendation by national kidney societies for glomerular filtration rate (eGFR) estimation in post-transplant setting. 99mTc-DTPA Technetium-99m Diethylene triamine penta acetic acid) renogram can identify early renal dysfunction much before ser...

Journal: :Iranian journal of immunology : IJI 2014
Sara Assadiasl Pedram Ahmadpoor Mohsen Nafar Mahboob Lessan Pezeshki Fateme Pourrezagholi Mahmoud Parvin Abtin Shahlaee Adel Sepanjnia Mohammad Hossein Nicknam Aliakbar Amirzargar

BACKGROUND Regulatory T cells have been suggested to have a protective role against acute rejection in allograft recipients. However, there is little information available about their contribution to chronic rejection process. The role of transforming growth factor-beta 1 (TGF-β1) as a profibrogenic and/or immunoregulatory cytokine in renal allografts is also controversial. OBJECTIVES To eval...

2014
Rubin Zhang

Allograft dysfunction is a common problem after kidney transplant. Allograft rejection is an important entity, and timely diagnosis and appropriate treatment are essential for caring transplant recipients. Hyperacute rejection is mediated by the preformed donor specific antibody, while accelerated acute rejection represents an anamnestic response by memory B and T cells. They occur early after ...

Journal: :International archives of allergy and immunology 2004
Stephan R Lederer Nicole Friedrich Rudolf Gruber Rüdiger Landgraf Marcel Toepfer Thomas Sitter

BACKGROUND The CD40-CD40L (CD154) costimulatory pathway plays a critical role in the pathogenesis of kidney allograft rejection. In renal transplant biopsies, CD4+CD40L+ graft-infiltrating cells were detected during chronic rejection in contrast to acute rejection episodes. Using a rapid noninvasive FACS procedure, we were able to demonstrate CD40L upregulation in peripheral blood of patients w...

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