نتایج جستجو برای: mad

تعداد نتایج: 3764  

Journal: :Tessera 1993

Journal: :Feminist Review 1991

Journal: :The Iowa Review 1976

Journal: :Journal of Literary and Cultural Disability Studies 2021

The article brings together the fields of mad studies (LeFrancois et al.), matricentric feminism (O’Reilly, Matricentric Feminism) and critical disability (Goodley, “Dis/entangling Critical Disability Studies”). aim is to expose challenge “relations ruling” (Smith 79) that both produce discipline “mad mothers disabled children.” analysis begins by exploring un/commonalities emerging histories t...

Journal: :Scandinavian Journal of Disability Research 2014

Journal: :Cancer research 1979
R G Parsons H D Todd R Kowal

A human DNA-binding protein, designated MAD-2, has recently been found to be elevated in the serum from patients with malignant diseases. MAD-2 has been purified approximately 500-fold from peritoneal and pleural fluids collected from cancer patients. Immunodiffusion studies have indicated that MAD-2 is immunochemically identical to human plasma fibronectin. The purified material has been resol...

Journal: :The European respiratory journal 2009
F Gagnadoux B Fleury B Vielle B Pételle N Meslier X L N'Guyen W Trzepizur J L Racineux

The aim of this study was to compare mandibular advancement device (MAd) therapy and continuous positive airway pressure (CPAP) for obstructive sleep apnoea/hypopnoea syndrome (OSAHS) after one-night polysomnographic (PSG) titration of both treatments. 59 OSAHS patients (apnoea/hypopnoea index (AHI): 34+/-13 events x h(-1); Epworth scale: 10.6+/-4.5) were included in a crossover trial of 8 week...

Journal: :Development 1996
S J Newfeld E H Chartoff J M Graff D A Melton W M Gelbart

The proteins necessary for signal transduction in cells responding to ligands of the TGF-beta family are largely unknown. We have previously identified Mad (Mothers against dpp), a gene that interacts with the TGF-beta family member encoded by decapentaplegic (dpp) in Drosophila. Assay of Mad's role in the DPP-dependent events of embryonic midgut development demonstrates that Mad is required fo...

1996
Stuart J. Newfeld Elena H. Chartoff Jonathan M. Graff Douglas A. Melton William M. Gelbart

The proteins necessary for signal transduction in cells responding to ligands of the TGF-β family are largely unknown. We have previously identified Mad (Mothers against dpp), a gene that interacts with the TGF-β family member encoded by decapentaplegic (dpp) in Drosophila. Assay of Mad’s role in the DPP-dependent events of embryonic midgut development demonstrates that Mad is required for any ...

Journal: :Hyperrhiz: New Media Cultures 2019

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