نتایج جستجو برای: fgfr

تعداد نتایج: 1529  

Journal: :Human molecular genetics 2016
Iva Gudernova Iva Vesela Lukas Balek Marcela Buchtova Hana Dosedelova Michaela Kunova Jakub Pivnicka Iva Jelinkova Lucie Roubalova Alois Kozubik Pavel Krejci

Activating mutations in the fibroblast growth factor receptor 3 (FGFR3) cause the most common genetic form of human dwarfism, achondroplasia (ACH). Small chemical inhibitors of FGFR tyrosine kinase activity are considered to be viable option for treating ACH, but little experimental evidence supports this claim. We evaluated five FGFR tyrosine kinase inhibitors (TKIs) (SU5402, PD173074, AZD1480...

2017
Shu Feng Longjiang Shao Patricia Castro Ilsa Coleman Peter S Nelson Paul D Smith Barry R Davies Michael Ittmann

Activation of the PI3K/AKT pathway occurs in the vast majority of advanced prostate cancers (PCas). Activation of fibroblast growth factor receptor (FGFR) signaling occurs in a wide variety of malignancies, including PCa. RNA-Seq of castration resistant PCa revealed expression of multiple FGFR signaling components compatible with FGFR signaling in all cases, with multiple FGF ligands expressed ...

Journal: :Folia histochemica et cytobiologica 2010
M Chrusciel R Rekawiecki A J Ziecik A Andronowska

The fibroblast growth factors (FGFs) are multifunctional proteins that, among other roles, regulate structural reorganization of uterine and placental vascular bed during pregnancy. Thus, we analyzed mRNA and protein expression and immunohistochemical localization of FGF-1 and FGF-2, and their receptors (FGFR-1 and FGFR-2) in the developing umbilical cord (UC) on days 40, 60, 75 and 90 of pregn...

Journal: :Blood 2002
Patricia E Burger Sandra Coetzee Wallace L McKeehan Mikio Kan Perry Cook Yong Fan Toshio Suda Robert P Hebbel Nicolas Novitzky William A Muller E Lynette Wilson

Recent experiments show that hematopoietic progenitor cell populations contain endothelial precursor cells. We have isolated a population of CD34(+) cells that expresses fibroblast growth factor receptor-1 (FGFR-1) and that differentiates into endothelial cells in vitro. We find that 4.5% +/- 2.1% of CD34(+) cells isolated from bone marrow, cord blood, and mobilized peripheral blood express FGF...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2015
Mehdi Touat Ecaterina Ileana Sophie Postel-Vinay Fabrice André Jean-Charles Soria

The fibroblast growth factor signaling pathway (FGFR signaling) is an evolutionary conserved signaling cascade that regulates several basic biologic processes, including tissue development, angiogenesis, and tissue regeneration. Substantial evidence indicates that aberrant FGFR signaling is involved in the pathogenesis of cancer. Recent developments of deep sequencing technologies have allowed ...

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2009
Alda Vidrich Jenny M Buzan Brooks Brodrick Chibuzo Ilo Leigh Bradley Kirstin Skaar Fendig Thomas Sturgill Steven M Cohn

Fibroblast growth factor receptor 3 (FGFR-3) is expressed in the lower crypt epithelium, where stem cells of the intestine reside. The role of FGFR-3 signaling in regulating features of intestinal morphogenesis was examined in FGFR-3-null (FGFR-3(-/-)) mice. FGFR-3(-/-) mice had only about half the number of intestinal crypts and a marked decrease in the number of functional clonogenic stem cel...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1999
G Venkataraman R Raman V Sasisekharan R Sasisekharan

Fibroblast growth factor (FGF) family plays key roles in development, wound healing, and angiogenesis. Understanding of the molecular nature of interactions of FGFs with their receptors (FGFRs) has been seriously limited by the absence of structural information on FGFR or FGF-FGFR complex. In this study, based on an exhaustive analysis of the primary sequences of the FGF family, we determined t...

Journal: :Cancer discovery 2013
Yi-Mi Wu Fengyun Su Shanker Kalyana-Sundaram Nickolay Khazanov Bushra Ateeq Xuhong Cao Robert J Lonigro Pankaj Vats Rui Wang Su-Fang Lin Ann-Joy Cheng Lakshmi P Kunju Javed Siddiqui Scott A Tomlins Peter Wyngaard Seth Sadis Sameek Roychowdhury Maha H Hussain Felix Y Feng Mark M Zalupski Moshe Talpaz Kenneth J Pienta Daniel R Rhodes Dan R Robinson Arul M Chinnaiyan

Through a prospective clinical sequencing program for advanced cancers, four index cases were identified which harbor gene rearrangements of FGFR2, including patients with cholangiocarcinoma, breast cancer, and prostate cancer. After extending our assessment of FGFR rearrangements across multiple tumor cohorts, we identified additional FGFR fusions with intact kinase domains in lung squamous ce...

Journal: :Molecular cell 2000
J Schlessinger A N Plotnikov O A Ibrahimi A V Eliseenkova B K Yeh A Yayon R J Linhardt M Mohammadi

The crystal structure of a dimeric 2:2:2 FGF:FGFR:heparin ternary complex at 3 A resolution has been determined. Within each 1:1 FGF:FGFR complex, heparin makes numerous contacts with both FGF and FGFR, thereby augmenting FGF-FGFR binding. Heparin also interacts with FGFR in the adjoining 1:1 FGF:FGFR complex to promote FGFR dimerization. The 6-O-sulfate group of heparin plays a pivotal role in...

Journal: :Cancer research 1993
M S Kobrin Y Yamanaka H Friess M E Lopez M Korc

Acidic and basic fibroblast growth factors are mitogenic polypeptides that are overexpressed in pancreatic cancer. To determine whether fibroblast growth factors may exert direct effects on pancreatic cancer cells in vivo, we compared the expression of the high-affinity type I fibroblast growth factor receptor (FGFR-1) in human pancreatic tissues. In the normal pancreas, FGFR-1 immunostaining w...

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