نتایج جستجو برای: friedreich

تعداد نتایج: 2912  

Journal: :Journal of Neurochemistry 2013

Journal: :Journal of neuropathology and experimental neurology 2013
Arnulf H Koeppen Joseph E Mazurkiewicz

Friedreich ataxia is an autosomal recessive disorder that affects children and young adults. The mutation consists of a homozygous guanine-adenine-adenine trinucleotide repeat expansion that causes deficiency of frataxin, a small nuclear genome-encoded mitochondrial protein. Low frataxin levels lead to insufficient biosynthesis of iron-sulfur clusters that are required for mitochondrial electro...

Journal: :Brain : a journal of neurology 2008
Michael C Fahey Phillip D Cremer Swee T Aw Lynette Millist Michael J Todd Owen B White Michael Halmagyi Louise A Corben Veronica Collins Andrew J Churchyard Kim Tan Lionel Kowal Martin B Delatycki

Friedreich ataxia (FRDA), the commonest of the inherited ataxias, is a multisystem neurodegenerative condition that affects ocular motor function. We assessed eye movement abnormalities in 20 individuals with genetically confirmed FRDA and compared these results to clinical measures. All subjects were assessed with infrared oculography. Fifteen individuals underwent a full protocol of eye movem...

Journal: :Journal of Neurochemistry 2013

Journal: :Pediatric Neurology Briefs 2002

Journal: :Journal of Child Neurology 2012

Journal: :International Journal of Clinical and Health Psychology 2018

Journal: :Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin 1882

2016
Yogesh K. Chutake Christina C. Lam Whitney N. Costello Michael P. Anderson Sanjay I. Bidichandani

Friedreich ataxia, the most prevalent inherited ataxia, is caused by an expanded GAA triplet-repeat sequence in intron 1 of the FXN gene. Repressive chromatin spreads from the expanded GAA triplet-repeat sequence to cause epigenetic silencing of the FXN promoter via altered nucleosomal positioning and reduced chromatin accessibility. Indeed, deficient transcriptional initiation is the predomina...

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