نتایج جستجو برای: aspartoacylase enzyme

تعداد نتایج: 241038  

2011
Sankar Surendran Srinivasagam Rajasankar

Aspartoacylase/aminoacylase II (ASPA/ACY II) is mainly synthesized in oligodendrocytes to contribute in myelin synthesis. Although axonal damage is seen in the brain with human immunodeficiency virus encephalitis (HIVE), ASPA contribution in the pathology is not known. Immunostaining study showed that ASPA protein is reduced in the white matter of patients with HIVE compared to the control. Wes...

2011
Nadine Mersmann Dmitri Tkachev Ruth Jelinek Philipp Thomas Röth Wiebke Möbius Torben Ruhwedel Sabine Rühle Wolfgang Weber-Fahr Alexander Sartorius Matthias Klugmann

Canavan Disease (CD) is a recessive leukodystrophy caused by loss of function mutations in the gene encoding aspartoacylase (ASPA), an oligodendrocyte-enriched enzyme that hydrolyses N-acetylaspartate (NAA) to acetate and aspartate. The neurological phenotypes of different rodent models of CD vary considerably. Here we report on a novel targeted aspa mouse mutant expressing the bacterial β-Gala...

Journal: :Proceedings for Annual Meeting of The Japanese Pharmacological Society 2019

Journal: :Journal of the neurological sciences 2016
Rebecca B Roscoe Christina Elliott Apostolos Zarros George S Baillie

Canavan disease (CD) is a rare leukodystrophy characterized by diffuse spongiform white matter degeneration, dysmyelination and intramyelinic oedema with consequent impairment of psychomotor development and early death. The molecular cause of CD has been identified as being mutations of the gene encoding the enzyme aspartoacylase (ASPA) leading to its functional deficiency. The physiological ro...

Journal: :Advances in experimental medicine and biology 2006
Reuben Matalon Kimberlee Michals-Matalon Sankar Surendran Stephen K Tyring

Canavan disease (CD) is an autosomal recessive disorder, characterized by spongy degeneration of the brain. Patients with CD have aspartoacylase (ASPA) deficiency, which results accumulation of N-acetylaspartic acid (NAA) in the brain and elevated excretion of urinary NAA. Clinically, patients with CD have macrocephaly, mental retardation and hypotonia. A knockout mouse for CD which was enginee...

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