apobec3g

نتایج جستجو برای: apobec3g

تعداد نتایج: 713 فیلتر نتایج به سال:

An intronic G run within HIV-1 intron 2 is critical for splicing regulation of vif mRNA.

Journal: :Journal of virology 2013

Marek Widera Steffen Erkelenz Frank Hillebrand Aikaterini Krikoni Darius Widera Wolfgang Kaisers René Deenen Michael Gombert Rafael Dellen Tanya Pfeiffer Barbara Kaltschmidt Carsten Münk Valerie Bosch Karl Köhrer Heiner Schaal

Within target T lymphocytes, human immunodeficiency virus type I (HIV-1) encounters the retroviral restriction factor APOBEC3G (apolipoprotein B mRNA-editing enzyme, catalytic polypeptide-like 3G; A3G), which is counteracted by the HIV-1 accessory protein Vif. Vif is encoded by intron-containing viral RNAs that are generated by splicing at 3' splice site (3'ss) A1 but lack splicing at 5'ss D2, ...

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An intronic G run within HIV-1 intron 2 is critical for splicing regulation of vif mRNA.

Inhibition of a NEDD8 Cascade Restores Restriction of HIV by APOBEC3G

T Cells Contain an RNase-Insensitive Inhibitor of APOBEC3G Deaminase Activity

Interactions of Murine APOBEC3 and Human APOBEC3G with Murine Leukemia Viruses

Hypermutation of hepatitis B virus genomes by APOBEC3G, APOBEC3C and APOBEC3H

Host APOBEC3G Protein Inhibits HCV Replication through Direct Binding at NS3

Expression and regulation of antiviral protein APOBEC3G in human neuronal cells

7SL RNA Mediates Virion Packaging of the Antiviral Cytidine Deaminase APOBEC3G

Deamination-Independent Inhibition of Hepatitis B Virus Reverse Transcription by APOBEC3G

Binding of RNA by APOBEC3G controls deamination-independent restriction of retroviruses