نتایج جستجو برای: soluble toll

تعداد نتایج: 112783  

2015
Dana C Blok Liesbeth M Kager Arie J Hoogendijk Ivar O Lede Wahid Rahman Rumana Afroz Paul Bresser Jaring S van der Zee Aniruddha Ghose Caroline E Visser Menno D de Jong Abu Shahed Md Zahed Md Anwar Husain Khan Mashrequl Alam Pravat Chandra Barua Mahtabuddin Hassan Ahmed Hossain Md Abu Tayab Rene Lutter Nick Day Arjen M Dondorp Alex F de Vos Cornelis van ’t Veer Tom van der Poll

BACKGROUND Tuberculosis (TB) is an important cause of morbidity and mortality worldwide. Toll-like-receptors (TLRs) are important for the recognition of the causative agent Mycobacterium tuberculosis. Negative regulation of TLRs is necessary to control deleterious inflammatory damage, but could provide a means of immune evasion by M. tuberculosis as well. METHODS To obtain insight in the exte...

2016
Fiona Long Yan Fong Pirkka V. Kirjavainen Hani El-Nezami

Lactobacillus rhamnosus GG (LGG) cells have been shown to promote type-1 immune responsiveness; however knowledge of immunomodulation of soluble factors secreted by LGG is limited. This is the first study to investigate whether LGG soluble factors promote a comparable immune responsiveness as the bacterial cells. Both treatments - LGG conditioned medium with (CM + LGG) or without (CM) LGG cells...

Journal: :gene, cell and tissue 0
seyed mohammad hashemi-shahri infectious diseases and tropical medicine research center, zahedan university of medical sciences, zahedan, ir iran mohsen taheri genetics of non-communicable disease research center, zahedan university of medical sciences, zahedan, ir iran; department of genetics, school of medicine, zahedan university of medical sciences, zahedan, ir iran; department of genetics, school of medicine, zahedan university of medical sciences, zahedan, ir iran. tel: +98-5413425793, fax: +98-5413425796 ali gadari infectious diseases and tropical medicine research center, zahedan university of medical sciences, zahedan, ir iran mohammad naderi infectious diseases and tropical medicine research center, zahedan university of medical sciences, zahedan, ir iran gholamreza bahari department of clinical biochemistry, school of medicine, zahedan university of medical sciences, zahedan, ir iran mohammad hashemi department of clinical biochemistry, school of medicine, zahedan university of medical sciences, zahedan, ir iran

conclusions tlr8 rs3764880 and tlr9 rs148805533 polymorphisms may not be risk factors for susceptibility to tuberculosis in a sample of iranian population. larger studies are required to validate our findings. results the allelic and genotypic frequencies of the tlr8 rs3764880 did not differ significantly between ptb and the controls. no significant difference was found between the groups regar...

Journal: :Infection and immunity 2010
Shih-Chin Cheng Louis Y A Chai Leo A B Joosten Anna Vecchiarelli Bernhard Hube Jos W Van Der Meer Bart Jan Kullberg Mihai G Netea

The innate immune system recognizes pathogen-associated molecular patterns (PAMPs) through pattern recognition receptors (PRR) and transduces downstream signaling to activate the host defense. Here we report that in addition to direct PAMP-PRR interactions, live Candida albicans cells can release soluble factors to actively potentiate interleukin-6 (IL-6) and IL-8 production induced in human mo...

2011
Marc Derive Sébastien Gibot

The triggering receptor expressed on myeloid cells-1 (TREM-1) is an immunoreceptor whose role is to amplify the inflammatory response mediated by the engagement of Toll-like and NOD-like receptors. As the expression of TREM-1 is believed to be upregulated during infection, this protein has been studied as a sepsis biomarker. In the previous issue of Critical Care, Su and colleagues reported on ...

Journal: :Blood 2010
Richard Frederick Ambinder

NK cells at different stages of maturation may help in tailoring optimal NK-cell responses in pathologic conditions, HSC transplantation, or vaccination. Along this line, it will be important to further identify cellular or soluble stimuli (eg, cytokines, Toll-like receptor ligands) capable of inducing rapid NK-cell differentiation and acquisition of the desired effector or regulatory function....

Journal: :Infection and immunity 2005
Roman Dziarski Dipika Gupta

Since the ability of peptidoglycan (PGN) to activate Toll-like receptor 2 (TLR2) was recently questioned, we reevaluated activation of TLR2 by PGN. Polymeric soluble or insoluble Staphylococcus aureus PGN, repurified by sodium dodecyl sulfate or phenol extraction, activated TLR2 at 0.1 to 1 or 10 mug/ml, respectively, and induced tumor necrosis factor alpha production. The TLR2 activation by PG...

Journal: :Blood 2004
Jérôme Pugin Sabine Stern-Voeffray Bruno Daubeuf Michael A Matthay Greg Elson Irène Dunn-Siegrist

In this paper, we show that plasma from patients with severe sepsis and septic shock but not normal plasma supports lipopolysaccharide (LPS) activation of epithelial cells expressing Toll-like receptor 4 (TLR4). Recombinant soluble myeloid differentiation protein-2 (MD-2) complemented normal plasma and allowed LPS activation of epithelial cells to levels measured with "septic" plasma, whereas s...

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