نتایج جستجو برای: ku70

تعداد نتایج: 763  

Journal: :Oncology reports 2015
Naoki Makita Itasu Ninomiya Tomoya Tsukada Koichi Okamoto Shinichi Harada Shinichi Nakanuma Seisho Sakai Isamu Makino Jun Kinoshita Hironori Hayashi Katsunobu Oyama Hisatoshi Nakagawara Tomoharu Miyashita Hidehiro Tajima Hiroyuki Takamura Sachio Fushida Tetsuo Ohta

Radiation therapy is one of the most promising therapeutic strategies in unresectable esophageal squamous cell carcinoma (ESCC). The histone deacetylase (HDAC) inhibitor has been shown to enhance radiosensitivity. Valproic acid (VPA) is a well-known drug used to treat seizure disorders and epilepsy, and has been shown to inhibit HDACs. We recently reported that a clinically safe dose of VPA enh...

2008
Zehui Hong Jie Jiang Li Lan Satoshi Nakajima Shin-ichiro Kanno Haruhiko Koseki Akira Yasui

DNA double-strand breaks (DSBs) represent the most toxic DNA damage arisen from endogenous and exogenous genotoxic stresses and are known to be repaired by either homologous recombination or nonhomologous end-joining processes. Although many proteins have been identified to participate in either of the processes, the whole processes still remain elusive. Polycomb group (PcG) proteins are epigen...

2003
Nicola Brady Terry J. Gaymes Manyee Cheung Ghulam J. Mufti Feyruz V. Rassool

Double strand breaks (DSBs) are considered the most lethal form of DNA damage for eukaryotic cells, and misrepair of DSB can cause cell death, chromosome instability, and cancer. Nonhomologous end-joining (NHEJ) is a major mechanism for the repair of DSBs. We previously reported that the cancer predisposition Bloom’s syndrome and myeloid leukemias demonstrate increased NHEJ activity and consequ...

Journal: :Stroke 2001
G W Kim N Noshita T Sugawara P H Chan

BACKGROUND AND PURPOSE Ku70 and Ku86, multifunctional DNA repair proteins, bind to broken DNA ends, including double-strand breaks, and trigger a DNA repair pathway. To investigate the involvement of these proteins in DNA fragmentation after ischemia/reperfusion, Ku protein expression was examined before and after transient focal cerebral ischemia (FCI) in mice. METHODS Adult male CD-1 mice w...

Journal: :Nucleic acids research 2004
Johanne Bentley Christine P Diggle Patricia Harnden Margaret A Knowles Anne E Kiltie

In human cells DNA double strand breaks (DSBs) can be repaired by the non-homologous end-joining (NHEJ) pathway. In a background of NHEJ deficiency, DSBs with mismatched ends can be joined by an error-prone mechanism involving joining between regions of nucleotide microhomology. The majority of joins formed from a DSB with partially incompatible 3' overhangs by cell-free extracts from human gli...

Journal: :Molecular and cellular biology 1999
B K Singleton M I Torres-Arzayus S T Rottinghaus G E Taccioli P A Jeggo

Ku is a heterodimeric protein with double-stranded DNA end-binding activity that operates in the process of nonhomologous end joining. Ku is thought to target the DNA-dependent protein kinase (DNA-PK) complex to the DNA and, when DNA bound, can interact and activate the DNA-PK catalytic subunit (DNA-PKcs). We have carried out a 3' deletion analysis of Ku80, the larger subunit of Ku, and shown t...

Journal: :Nucleic acids research 2016
Praveen L Patidar Edward A Motea Farjana J Fattah Yunyun Zhou Julio C Morales Yang Xie Harold R Garner David A Boothman

Ku70-binding protein 5 (Kub5)-Hera (K-H)/RPRD1B maintains genetic integrity by concomitantly minimizing persistent R-loops and promoting repair of DNA double strand breaks (DSBs). We used tandem affinity purification-mass spectrometry, co-immunoprecipitation and gel-filtration chromatography to define higher-order protein complexes containing K-H scaffolding protein to gain insight into its cel...

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