نتایج جستجو برای: apobec3g

تعداد نتایج: 713  

2016
Pierre-Jean Racine Célia Chamontin Hugues de Rocquigny Serena Bernacchi Jean-Christophe Paillart Marylène Mougel

HIV-1 is a retrovirus replicating within cells by reverse transcribing its genomic RNA (gRNA) into DNA. Within cells, virus assembly requires the structural Gag proteins with few accessory proteins, notably the viral infectivity factor (Vif) and two copies of gRNA as well as cellular factors to converge to the plasma membrane. In this process, the nucleocapsid (NC) domain of Gag binds to the pa...

Journal: :Nucleic Acids Research 2006
Stefán R. Jónsson Guylaine Haché Mark D. Stenglein Scott C. Fahrenkrug Valgerdur Andrésdóttir Reuben S. Harris

The APOBEC3 proteins are unique to mammals. Many inhibit retrovirus infection through a cDNA cytosine deamination mechanism. HIV-1 neutralizes this host defense through Vif, which triggers APOBEC3 ubiquitination and degradation. Here, we report an APOBEC3F-like, double deaminase domain protein from three artiodactyls: cattle, pigs and sheep. Like their human counterparts, APOBEC3F and APOBEC3G,...

Journal: :The Journal of clinical investigation 2011
Qingqing Ding Chun-Ju Chang Xiaoming Xie Weiya Xia Jer-Yen Yang Shao-Chun Wang Yan Wang Jiahong Xia Libo Chen Changchun Cai Huabin Li Chia-Jui Yen Hsu-Ping Kuo Dung-Fang Lee Jingyu Lang Longfei Huo Xiaoyun Cheng Yun-Ju Chen Chia-Wei Li Long-Bin Jeng Jennifer L Hsu Long-Yuan Li Alai Tan Steven A Curley Lee M Ellis Raymond N Dubois Mien-Chie Hung

Colorectal cancer is the second leading cause of death from cancer in the United States. Metastases in the liver, the most common metastatic site for colorectal cancer, are found in one-third of the patients who die of colorectal cancer. Currently, the genes and molecular mechanisms that are functionally critical in modulating colorectal cancer hepatic metastasis remain unclear. Here, we report...

Journal: :PLoS Pathogens 2007
Vanessa B Soros Wes Yonemoto Warner C Greene

APOBEC3G (A3G) is a potent antiretroviral deoxycytidine deaminase that, when incorporated into HIV virions, hypermutates nascent viral DNA formed during reverse transcription. HIV Vif counters the effect of A3G by depleting intracellular stores of the enzyme, thereby blocking its virion incorporation. Through pulse-chase analyses, we demonstrate that virion A3G is mainly recruited from the cell...

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