نتایج جستجو برای: onset diabetes of the young mody

تعداد نتایج: 22867488  

Journal: :Endocrinologia y nutricion : organo de la Sociedad Espanola de Endocrinologia y Nutricion 2009
Martine Vaxillaire Philippe Froguel

There are several monogenic disorders of pancreatic β-cell function, characterized by various degrees of chronic hyperglycemia. They are usually diagnosed early in life, in neonates or during infancy, in childhood and even in young adulthood1-3. The identification of causal mutations in a dozen of different genes has already proven to have a great clinical impact opening new avenues in genomic ...

2016
Alla K. Ovsyannikova Oksana D. Rymar Elena V. Shakhtshneider Vadim V. Klimontov Elena A. Koroleva Natalya E. Myakina Mikhail I. Voevoda

Maturity-onset diabetes of the young (MODY) is a heterogeneous group of diseases associated with gene mutations leading to dysfunction of pancreatic β-cells. Thirteen identified MODY variants differ from each other by the clinical course and treatment requirement. Currently, MODY subtypes 1-5 are best-studied, descriptions of the other forms are sporadic. This article reports a MODY12 clinical ...

2017
Anna Wędrychowicz Ewa Tobór Magdalena Wilk Ewa Ziółkowska-Ledwith Anna Rams Katarzyna Wzorek Barbara Sabal Małgorzata Stelmach Jerzy B. Starzyk

OBJECTIVE The aim of the study was to evaluate the clinical phenotypes of glucokinase-maturity-onset diabetes of the young (GCK-MODY) pediatric patients from Southwest Poland and to search for phenotype-genotype correlations. METHODS We conducted a retrospective analysis of data on 37 CGK-MODY patients consisting of 21 girls and 16 boys of ages 1.9-20.1 (mean 12.5±5.2) years, treated in our c...

2010
Hana Lango Allen Stefan Johansson Sian Ellard Beverley Shields Jens K. Hertel Helge Ræder Kevin Colclough Anders Molven Timothy M. Frayling Pål R. Njølstad Andrew T. Hattersley Michael N. Weedon

OBJECTIVE Mutations in the HNF1A gene are the most common cause of maturity-onset diabetes of the young (MODY). There is a substantial variation in the age at diabetes diagnosis, even within families where diabetes is caused by the same mutation. We investigated the hypothesis that common polygenic variants that predispose to type 2 diabetes might account for the difference in age at diagnosis....

Journal: :Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy 2019

Journal: :PLoS Biology 2005
Haitao Yang Weiqing Xie Xiaoyu Xue Kailin Yang Jing Ma Wenxue Liang Qi Zhao Zhe Zhou Duanqing Pei John Ziebuhr Rolf Hilgenfeld Kwok Yung Yuen Luet Wong Guangxia Gao Saijuan Chen Zhu Chen Dawei Ma Mark Bartlam Zihe Rao

Macrosomia is associated with considerable neonatal and maternal morbidity. Factors that predict macrosomia are poorly understood. The increased rate of macrosomia in the offspring of pregnant women with diabetes and in congenital hyperinsulinaemia is mediated by increased foetal insulin secretion. We assessed the in utero and neonatal role of two key regulators of pancreatic insulin secretion ...

Journal: :The Journal of clinical investigation 1999
W M Macfarlane T M Frayling S Ellard J C Evans L I Allen M P Bulman S Ayres M Shepherd P Clark A Millward A Demaine T Wilkin K Docherty A T Hattersley

The transcription factor insulin promoter factor-1 (IPF-1) plays a central role in both the development of the pancreas and the regulation of insulin gene expression in the mature pancreatic beta cell. A dominant-negative frameshift mutation in the IPF-l gene was identified in a single family and shown to cause pancreatic agenesis when homozygous and maturity-onset diabetes of the young (MODY) ...

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