نتایج جستجو برای: fgfr

تعداد نتایج: 1529  

Journal: :Oncology reports 2007
Koichi Sugiura Soji Ozawa Yuko Kitagawa Masakazu Ueda Masaki Kitajima

Overexpression of aFGF, bFGF and FGFR-1 has been reported in various cancers, and it has been suggested that it may be a poor prognostic factor in cases with solid tumors. Therefore, we attempted to determine whether overexpression of aFGF, bFGF and FGFR-1 might also be a poor prognostic factor in patients with esophageal squamous cell carcinoma, and examined the expression of aFGF, bFGF and FG...

Journal: :Cell 1996
Chuxia Deng Anthony Wynshaw-Boris Fen Zhou Ann Kuo Philip Leder

Endochondral ossification is a major mode of bone that occurs as chondrocytes undergo proliferation, hypertrophy, cell death, and osteoblastic replacement. We have identified a role for fibroblast growth factor receptor 3 (FGFR-3) in this process by disrupting the murine Fgfr-3 gene to produce severe and progressive bone dysplasia with enhanced and prolonged endochondral bone growth. This growt...

Journal: :Cancer discovery 2013
Maria Vittoria Dieci Monica Arnedos Fabrice Andre Jean Charles Soria

The fibroblast growth factor/fibroblast growth factor receptor (FGF/FGFR) signaling pathway plays a fundamental role in many physiologic processes, including embryogenesis, adult tissue homeostasis, and wound healing, by orchestrating angiogenesis. Ligand-independent and ligand-dependent activation have been implicated in a broad range of human malignancies and promote cancer progression in tum...

Journal: :Journal of cell science 2004
Peetra Magnusson Charlotte Rolny Lars Jakobsson Charlotte Wikner Yan Wu Daniel J Hicklin Lena Claesson-Welsh

We have employed embryoid bodies derived from murine embryonal stem cells to study effects on vascular development induced by fibroblast growth factor (FGF)-2 and FGF receptor-1, in comparison to the established angiogenic factor vascular endothelial growth factor (VEGF)-A and its receptor VEGF receptor-2. Exogenous FGF-2 promoted formation of morphologically distinct, long slender vessels in t...

Journal: :Cancer discovery 2017
Lipika Goyal Supriya K Saha Leah Y Liu Giulia Siravegna Ignaty Leshchiner Leanne G Ahronian Jochen K Lennerz Phuong Vu Vikram Deshpande Avinash Kambadakone Benedetta Mussolin Stephanie Reyes Laura Henderson Jiaoyuan Elisabeth Sun Emily E Van Seventer Joseph M Gurski Sabrina Baltschukat Barbara Schacher-Engstler Louise Barys Christelle Stamm Pascal Furet David P Ryan James R Stone A John Iafrate Gad Getz Diana Graus Porta Ralph Tiedt Alberto Bardelli Dejan Juric Ryan B Corcoran Nabeel Bardeesy Andrew X Zhu

Genetic alterations in the fibroblast growth factor receptor (FGFR) pathway are promising therapeutic targets in many cancers, including intrahepatic cholangiocarcinoma (ICC). The FGFR inhibitor BGJ398 displayed encouraging efficacy in patients with FGFR2 fusion-positive ICC in a phase II trial, but the durability of response was limited in some patients. Here, we report the molecular basis for...

Journal: :Development 2004
Stefanie Sudhop Francois Coulier Annette Bieller Angelika Vogt Tobias Hotz Monika Hassel

Signalling through fibroblast growth factors (FGFR) is essential for proper morphogenesis in higher evolved triploblastic organisms. By screening for genes induced during morphogenesis in the diploblastic Hydra, we identified a receptor tyrosine kinase (kringelchen) with high similarity to FGFR tyrosine kinases. The gene is dynamically upregulated during budding, the asexual propagation of Hydr...

Journal: :Genes to cells : devoted to molecular & cellular mechanisms 2011
Tokiharu Sato Anir Enkhbat Katsuji Yoshioka

We previously reported that the scaffold protein c-Jun NH₂-terminal kinase (JNK)/stress-activated protein kinase-associated protein 1 (JSAP1) functions in cerebellar granule cell precursors (GCPs) to promote their cell-cycle exit and differentiation. In this study, we used immunocytochemistry to examine the subcellular distribution of JSAP1 in proliferating cultured GCPs. We found that when sti...

2012
Eleonora Brunello Matteo Brunelli Giuseppe Bogina Anna Caliò Erminia Manfrin Alessia Nottegar Marco Vergine Annamaria Molino Emilio Bria Francesco Massari Giampaolo Tortora Sara Cingarlini Serena Pedron Marco Chilosi Giuseppe Zamboni Keith Miller Guido Martignoni Franco Bonetti

BACKGROUND Lobular breast carcinoma usually shows poor responsiveness to chemotherapies and often lacks targeted therapies. Since FGFR1 expression has been shown to play pivotal roles in primary breast cancer tumorigenesis, we sought to analyze the status of FGFR1 gene in a metastatic setting of lobular breast carcinoma, since promising FGFR1 inhibitors has been recently developed. METHODS Fi...

2011
Marianne E. Marshall Trista K. Hinz Scott A. Kono Katherine R. Singleton Brady Bichon Kathryn E. Ware Lindsay Marek Barbara A. Frederick David Raben Lynn E. Heasley

Purpose: We previously reported that a fibroblast growth factor (FGF) receptor (FGFR) signaling pathway drives growth of lung cancer cell lines of squamous and large cell histologies. Herein, we explored FGFR dependency in cell lines derived from the tobacco-related malignancy, head and neck squamous cell

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2016
Koos Koole Diede Brunen Pauline M W van Kempen Rob Noorlag Remco de Bree Cor Lieftink Robert J J van Es René Bernards Stefan M Willems

PURPOSE FGFR1 is a promising therapeutic target in multiple types of solid tumors, including head and neck squamous cell carcinoma (HNSCC). FGFR inhibitors have shown great therapeutic value in preclinical models. However, resistance remains a major setback. In this study, we have investigated the prognostic value of FGFR1 expression in HNSCC, the therapeutic relevance of targeting FGFR with AZ...

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