نتایج جستجو برای: replication efficiency

تعداد نتایج: 486750  

2011
Bifeng Yuan Jianshuang Wang Huachuan Cao Ruobai Sun Yinsheng Wang

Human cells are constantly exposed to environmental and endogenous agents which can induce damage to DNA. Understanding the implications of these DNA modifications in the etiology of human diseases requires the examination about how these DNA lesions block DNA replication and induce mutations in cells. All previously reported shuttle vector-based methods for investigating the cytotoxic and muta...

Journal: :Journal of virology 1999
P J Wilderman B Hu M E Woodworth

Simian virus 40 (SV40) is an excellent model system for investigating the cis- and trans-acting factors involved in eukaryotic DNA replication because it uses host enzymes, with the exception of the virus-encoded T-antigen (T-ag), to replicate its genome. Although its origin of replication (ori) is essential for DNA replication, there are transcriptional promoters and enhancers that affect DNA ...

Journal: :Journal of immunology 1999
R Wagner B Leschonsky E Harrer C Paulus C Weber B D Walker S Buchbinder H Wolf J R Kalden T Harrer

It has been hypothesized that sequence variation within CTL epitopes leading to immune escape plays a role in the progression of HIV-1 infection. Only very limited data exist that address the influence of biologic characteristics of CTL epitopes on the emergence of immune escape variants and the efficiency of suppression HIV-1 by CTL. In this report, we studied the effects of HIV-1 CTL epitope ...

2014
Christopher D. Richardson Joachim J. Li

Eukaryotic cells must inhibit re-initiation of DNA replication at each of the thousands of origins in their genome because re-initiation can generate genomic alterations with extraordinary frequency. To minimize the probability of re-initiation from so many origins, cells use a battery of regulatory mechanisms that reduce the activity of replication initiation proteins. Given the global nature ...

Journal: :Journal of virology 2012
Cristina Capella Michael-John Beltejar Caitlin Brown Vincent Fong Waaqo Daddacha Baek Kim Stephen Dewhurst

Mutations that reduce the efficiency of deoxynucleoside (dN) triphosphate (dNTP) substrate utilization by the HIV-1 DNA polymerase prevent viral replication in resting cells, which contain low dNTP concentrations, but not in rapidly dividing cells such as cancer cells, which contain high levels of dNTPs. We therefore tested whether mutations in regions of the adenovirus type 5 (Ad5) DNA polymer...

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