نتایج جستجو برای: neurodegeneration with brain iron accumulation

تعداد نتایج: 9480785  

Pantothenate kinase- associated neurodegeneration (PKAN) syndrome is a rare autosomal recessive disorder characterized by progressive extrapyramidal dysfunction and iron accumulation in the brain and axonal spheroids in the central nervous system. It has been shown that the disorder is caused by mutations in PANK2 gene which codes for a mitochondrial enzyme participating in coenzyme A biosynthe...

Journal: :Brain research 2003
Colette Grabill Afonso C Silva Sophia S Smith Alan P Koretsky Tracey A Rouault

Genetic ablation of iron regulatory protein 2 (IRP-2), a protein responsible for post-transcriptional regulation of expression of several iron metabolism proteins, predisposes IRP-2 -/- mice to develop adult onset neurodegenerative disease. Ferric iron reproducibly accumulates within axonal tracts and neuronal cell bodies in discrete regions of the brain, and areas of iron accumulation colocali...

2018
Christine Kaindlstorfer Kurt A. Jellinger Sabine Eschlböck Nadia Stefanova Günter Weiss Gregor K. Wenning

Iron is essential for cellular development and maintenance of multiple physiological processes in the central nervous system. The disturbance of its homeostasis leads to abnormal iron deposition in the brain and causes neurotoxicity via generation of free radicals and oxidative stress. Iron toxicity has been established in the pathogenesis of Parkinson's disease; however, its contribution to mu...

Journal: :International Journal of Research in Medical Sciences 2014

Journal: :Oman medical journal 2017
Nabil Al Macki Ismail Al Rashdi

Mutations in the C19orf12 gene are known to cause mitochondrial membrane protein-associated neurodegeneration (MPAN), which is a neurodegeneration with brain iron accumulation (NBIA) type 4 disorder. To the best of our knowledge, this is the first report of a genetically confirmed case of MPAN from Oman. A novel homozygous deletion of exon 2 of the C19orf12 gene was confirmed on the proband, a ...

Journal: :Neurology 2008
A Gregory S K Westaway I E Holm P T Kotzbauer P Hogarth S Sonek J C Coryell T M Nguyen N Nardocci G Zorzi D Rodriguez I Desguerre E Bertini A Simonati B Levinson C Dias C Barbot I Carrilho M Santos I Malik J Gitschier S J Hayflick

OBJECTIVE Mutations in the gene encoding phospholipase A(2) group VI (PLA2G6) are associated with two childhood neurologic disorders: infantile neuroaxonal dystrophy (INAD) and idiopathic neurodegeneration with brain iron accumulation (NBIA). INAD is a severe progressive psychomotor disorder in which axonal spheroids are found in brain, spinal cord, and peripheral nerves. High globus pallidus i...

Journal: :AJNR. American journal of neuroradiology 2006
S J Hayflick M Hartman J Coryell J Gitschier H Rowley

BACKGROUND AND OBJECTIVE Patients with a clinical diagnosis of neurodegeneration with brain iron accumulation (NBIA, formerly called Hallervorden-Spatz syndrome) often have mutations in PANK2, the gene encoding pantothenate kinase 2. We investigated correlations between brain MR imaging changes, mutation status, and clinical disease features. METHODS Brain MRIs from patients with NBIA were re...

2018
Alan Zanardi Antonio Conti Marco Cremonesi Patrizia D'Adamo Enrica Gilberti Pietro Apostoli Carlo Vittorio Cannistraci Alberto Piperno Samuel David Massimo Alessio

Aceruloplasminemia is a monogenic disease caused by mutations in the ceruloplasmin gene that result in loss of protein ferroxidase activity. Ceruloplasmin plays a role in iron homeostasis, and its activity impairment leads to iron accumulation in liver, pancreas, and brain. Iron deposition promotes diabetes, retinal degeneration, and progressive neurodegeneration. Current therapies mainly based...

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