نتایج جستجو برای: gliosis

تعداد نتایج: 2309  

2011
Marina Martinez Jean-Michel Brezun Christian Xerri

Sensorimotor activity has been shown to play a key role in functional outcome after extensive brain damage. This study was aimed at assessing the influence of sensorimotor experience through subject-environment interactions on the time course of both lesion and gliosis volumes as well as on the recovery of forelimb sensorimotor abilities following focal cortical injury. The lesion consisted of ...

Journal: :Investigative ophthalmology & visual science 2004
Félix Vázquez-Chona Bong K Song Eldon E Geisert

PURPOSE The goal of this study was to define the temporal changes in gene expression after retinal injury and to relate these changes to the inflammatory and reactive response. A specific emphasis was placed on the tetraspanin family of proteins and their relationship with markers of reactive gliosis. METHODS Retinal tears were induced in adult rats by scraping the retina with a needle. After...

Journal: :Vision Research 2015
Qian Jian Zui Tao Yaochen Li Zheng Qin Yin

Our aim is to define related molecular events on how dormant Müller glia cells re-enter the cell cycle, proliferate and produce new retinal neurons from initial injury to glial scar formation. Sodium iodate (NaIO3) was used to induce acute retinal injury. Long-Evans rats were administered with NaIO3 or phosphate-buffered saline by intraperitoneal injection. The proliferation, dedifferentiation ...

2010
Wendy M. Aartsen Koen W. R. van Cleef Lucie P. Pellissier Robert M. Hoek Rogier M. Vos Bas Blits Erich M. E. Ehlert Kamaljit S. Balaggan Robin R. Ali Joost Verhaagen Jan Wijnholds

BACKGROUND Müller cell gliosis occurs in various retinal pathologies regardless of the underlying cellular defect. Because activated Müller glial cells span the entire retina and align areas of injury, they are ideal targets for therapeutic strategies, including gene therapy. METHODOLOGY/PRINCIPAL FINDINGS We used adeno-associated viral AAV2/6 vectors to transduce mouse retinas. The transduct...

2017
Ji Hyeon Ahn Myoung Cheol Shin Joon Ha Park In Hye Kim Jeong-Hwi Cho Tae-Kyeong Lee Jae-Chul Lee Bai Hui Chen Bich Na Shin Hyun-Jin Tae Jinseu Park Soo Young Choi Yun Lyul Lee Dae Won Kim Yang Hee Kim Moo-Ho Won Jun Hwi Cho

Therapeutic exercise is an integral component of the rehabilitation of patients who have suffered a stroke. The objective of the present study was to use immunohistochemistry to investigate the effects of post‑ischemic exercise on neuronal damage or death and gliosis in the aged gerbil hippocampus following transient cerebral ischemia. Aged gerbils (male; age, 22‑24 months) underwent ischemia a...

2017
Tzu-Hsien Tsai Cheng-Jei Lin Sarah Chua Sheng-Ying Chung Cheng-Hsu Yang Meng-Shen Tong Chi-Ling Hang

BACKGROUND Vascular cognitive impairment (VCI) is a spectrum of cognitive impairment caused by various chronic diseases including aging, hypertension, and diabetes mellitus. Oxidative and inflammatory reactions induced by chronic cerebral hypoperfusion (CHP) are believed to cause VCI. Melatonin is reported to possess anti-oxidation and anti-inflammation effects. This study was designed to inves...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 1999
R J McKeon M J Jurynec C R Buck

Chondroitin sulfate proteoglycans (CS-PGs) expressed by reactive astrocytes may contribute to the axon growth-inhibitory environment of the injured CNS. The specific potentially inhibitory CS-PGs present in areas of reactive gliosis, however, have yet to be thoroughly examined. In this study, we used immunohistochemistry, combined immunohistochemistry-in situ hybridization, immunoblot analysis,...

2013
Juan Qu Tatjana C. Jakobs

Reactive gliosis is a complex process that involves changes in gene expression and morphological remodeling. The mouse optic nerve, where astrocytes, microglia and oligodendrocytes interact with retinal ganglion cell axons and each other, is a particularly suitable model for studying the molecular mechanisms of reactive gliosis. We triggered gliosis at the mouse optic nerve head by retro orbita...

Journal: :Arquivos de neuro-psiquiatria 2007
Cristiane Low Eliana Garzon Henrique Carrete Luis Celso Vilanova Elza Márcia T Yacubian Américo C Sakamoto

OBJECTIVE Early brain insults can cause cavitary lesions including porencephaly (POR) and multicystic encephalopathy (MCE). The objective of this study was to investigate clinical and electrographic correlates associated to these types of destructive brain lesions. METHOD Patients with POR and MCE were selected and submitted to clinical and Video-EEG monitoring. The following variables were a...

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