Considerations regarding the etiology of hemolytic jaundice are inextricably bound up with the presence of spherocytosis and the associated increase in hypotonic saline fragility, and the effect of splenectomy in the amelioration of this disorder. Although many different opinions are advanced regarding the etiology of the disease, the outstanding ones are those postulating a faulty bone marrow ...

Objective(s): Neurotrophic factors secreting cells (NTS-SCs) may be a superior cell source for cell-based therapy in neurodegenerative diseases. NTS-SCs are able to secrete some neurotrophic Such as nerve growth factor and glia-derived neurotrophic factor. Our primary aim was to assess transplantation of neurotrophic factor secreting cells derived from human adipose-derived stem cells (hADSCs) ...

Glycerophosphatide phosphorusis determined in ether-methanol extracts of blood serum by a nondigestive procedure which utilizes a sulfuric-periodic acid reagent. Sphingomyelinphosphorusis not liberated. The method shows good agreement with the sum of lecithin, cephalin, and lysolecithin phosphorus when the lipids are separated by thin-layer chromatography. Lecithin in an artificial lipid mixtur...

The acyl group of acyl dihydroxyacetone phosphate was shown to be enzymatically transferred in guinea pig liver mitochondria to various acceptors such as lysolecithin, lysophosphatidyl ethanolamine and sn-glycerol3-phosphate to form lecithin, phosphatidyl ethanolamine and phosphatidate, respectively. Coenzyme A and Mg*, but not ATP, were required for this reaction. A rapid exchange of acyl grou...

Buffalo spermatozoa have been studied cytochemically and biochemically to investigate their phospholipids, which appear to be phosphatidyl choline, lysolecithin, phosphatidyl ethanolamine, phosphatidyl inositol, phosphatidyl glycerol and monogalactosyl diglyceride. Most of them form lipoprotein complexes in the regions of the plasma membrane, the post-nuclear cap and the mid-piece. The lipid co...

Multiple sclerosis is an inflammatory demyelinating disease of the central nervous system characterized by plaque formation containing lost oligodendrocytes, myelin, axons, and neurons. Remyelination is an endogenous repair mechanism whereby new myelin is produced subsequent to proliferation, recruitment, and differentiation of oligodendrocyte precursor cells into myelin-forming oligodendrocyte...