Early Marjolin's Ulcer after Minimal Superficial Burn
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Early Marjolin’s Ulcer after Minimal Superficial Burn
Dear Editor, Marjolin’s ulcer is a rare, well-defined, uncommon, and often aggressive malignant transformation,1 secondary to burn injuries and other inflammatory changes such as venous insufficiency ulcers, pressure ulcers, traumatic wounds, cystostomy sites, scarring from lupus, amputation stumps, chronic lymphedema, chronic pilonidal sinuses, hidradenitis suppurativa, chronic ulcers of lepro...
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Background & Objective: Chronic non healing ulcers or scar undergoing malignant transformation is a rare phenomenon with an estimated incidence of 2%. Though, numerous predisposing factors have been identified, still it is rarely diagnosed and commonly mistaken for a benign condition. The objective of this study was to verify the efficacy of the Fine Needle Aspiration Cytology/ Scrape cytology ...
full textSpectrum of Malignant Transformation in Marjolins Ulcer: Our Experience of This Rare Phenomenon
Background & Objective: Chronic non healing ulcers or scar undergoing malignant transformation is a rare phenomenon with an estimated incidence of 2%. Though, numerous predisposing factors have been identified, still it is rarely diagnosed and commonly mistaken for a benign condition. The objective of this study was to verify the efficacy of the Fine Needle Aspiration Cytology/ Scrape cytology ...
full textSpectrum of Malignant Transformation in Marjolins Ulcer: Our Experience of This Rare Phenomenon
Background & Objective: Chronic non healing ulcers or scar undergoing malignant transformation is a rare phenomenon with an estimated incidence of 2%. Though, numerous predisposing factors have been identified, still it is rarely diagnosed and commonly mistaken for a benign condition. The objective of this study was to verify the efficacy of the Fine Needle Aspiration Cytology/ Scrape cytolo...
full textGlucocorticoid-induced, caspase-dependent organ apoptosis early after burn injury.
Immune suppression and increased apoptotic loss of circulating lymphocytes have been reported after burn injury. However, little is known about the underlying mechanisms responsible for the increased apoptosis of lymphoid and parenchymal cells in solid organs and the role played by inflammatory mediators, such as tumor necrosis factor-alpha (TNF-alpha) and Fas ligand (FasL), as well as by gluco...
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Journal title
volume 38 issue 1
pages 69- 70
publication date 2013-03-01
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