Mapping autism risk loci using genetic linkage and chromosomal rearrangements.

نویسندگان

  • Peter Szatmari
  • Andrew D Paterson
  • Lonnie Zwaigenbaum
  • Wendy Roberts
  • Jessica Brian
  • Xiao-Qing Liu
  • John B Vincent
  • Jennifer L Skaug
  • Ann P Thompson
  • Lili Senman
  • Lars Feuk
  • Cheng Qian
  • Susan E Bryson
  • Marshall B Jones
  • Christian R Marshall
  • Stephen W Scherer
  • Veronica J Vieland
  • Christopher Bartlett
  • La Vonne Mangin
  • Rhinda Goedken
  • Alberto Segre
  • Margaret A Pericak-Vance
  • Michael L Cuccaro
  • John R Gilbert
  • Harry H Wright
  • Ruth K Abramson
  • Catalina Betancur
  • Thomas Bourgeron
  • Christopher Gillberg
  • Marion Leboyer
  • Joseph D Buxbaum
  • Kenneth L Davis
  • Eric Hollander
  • Jeremy M Silverman
  • Joachim Hallmayer
  • Linda Lotspeich
  • James S Sutcliffe
  • Jonathan L Haines
  • Susan E Folstein
  • Joseph Piven
  • Thomas H Wassink
  • Val Sheffield
  • Daniel H Geschwind
  • Maja Bucan
  • W Ted Brown
  • Rita M Cantor
  • John N Constantino
  • T Conrad Gilliam
  • Martha Herbert
  • Clara Lajonchere
  • David H Ledbetter
  • Christa Lese-Martin
  • Janet Miller
  • Stan Nelson
  • Carol A Samango-Sprouse
  • Sarah Spence
  • Matthew State
  • Rudolph E Tanzi
  • Hilary Coon
  • Geraldine Dawson
  • Bernie Devlin
  • Annette Estes
  • Pamela Flodman
  • Lambertus Klei
  • William M McMahon
  • Nancy Minshew
  • Jeff Munson
  • Elena Korvatska
  • Patricia M Rodier
  • Gerard D Schellenberg
  • Moyra Smith
  • M Anne Spence
  • Chris Stodgell
  • Ping Guo Tepper
  • Ellen M Wijsman
  • Chang-En Yu
  • Bernadette Rogé
  • Carine Mantoulan
  • Kerstin Wittemeyer
  • Annemarie Poustka
  • Bärbel Felder
  • Sabine M Klauck
  • Claudia Schuster
  • Fritz Poustka
  • Sven Bölte
  • Sabine Feineis-Matthews
  • Evelyn Herbrecht
  • Gabi Schmötzer
  • John Tsiantis
  • Katerina Papanikolaou
  • Elena Maestrini
  • Elena Bacchelli
  • Francesca Blasi
  • Simona Carone
  • Claudio Toma
  • Herman Van Engeland
  • Maretha de Jonge
  • Chantal Kemner
  • Frederieke Koop
  • Marjolein Langemeijer
  • Channa Hijmans
  • Wouter G Staal
  • Gillian Baird
  • Patrick F Bolton
  • Michael L Rutter
  • Emma Weisblatt
  • Jonathan Green
  • Catherine Aldred
  • Julie-Anne Wilkinson
  • Andrew Pickles
  • Ann Le Couteur
  • Tom Berney
  • Helen McConachie
  • Anthony J Bailey
  • Kostas Francis
  • Gemma Honeyman
  • Aislinn Hutchinson
  • Jeremy R Parr
  • Simon Wallace
  • Anthony P Monaco
  • Gabrielle Barnby
  • Kazuhiro Kobayashi
  • Janine A Lamb
  • Ines Sousa
  • Nuala Sykes
  • Edwin H Cook
  • Stephen J Guter
  • Bennett L Leventhal
  • Jeff Salt
  • Catherine Lord
  • Christina Corsello
  • Vanessa Hus
  • Daniel E Weeks
  • Fred Volkmar
  • Maïté Tauber
  • Eric Fombonne
  • Andy Shih
  • Kacie J Meyer
چکیده

Autism spectrum disorders (ASDs) are common, heritable neurodevelopmental conditions. The genetic architecture of ASDs is complex, requiring large samples to overcome heterogeneity. Here we broaden coverage and sample size relative to other studies of ASDs by using Affymetrix 10K SNP arrays and 1,181 [corrected] families with at least two affected individuals, performing the largest linkage scan to date while also analyzing copy number variation in these families. Linkage and copy number variation analyses implicate chromosome 11p12-p13 and neurexins, respectively, among other candidate loci. Neurexins team with previously implicated neuroligins for glutamatergic synaptogenesis, highlighting glutamate-related genes as promising candidates for contributing to ASDs.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Mapping autism risk loci using genetic linkage and chromosomal rearrangements

Autism spectrum disorders (ASDs) are common, heritable neurodevelopmental conditions. The genetic architecture of ASDs is complex, requiring large samples to overcome heterogeneity. Here we broaden coverage and sample size relative to other studies of ASDs by using Affymetrix 10K SNP arrays and 1,168 families with at least two affected individuals, performing the largest linkage scan to date wh...

متن کامل

Mapping autism risk loci using genetic linkage and chromosomal rearrangements

Autism spectrum disorders (ASDs) are common, heritable neurodevelopmental conditions. The genetic architecture of ASDs is complex, requiring large samples to overcome heterogeneity. Here we broaden coverage and sample size relative to other studies of ASDs by using Affymetrix 10K SNP arrays and 1,168 families with at least two affected individuals, performing the largest linkage scan to date wh...

متن کامل

Mapping autism risk loci using genetic linkage and chromosomal rearrangements

Autism spectrum disorders (ASDs) are common, heritable neurodevelopmental conditions. The genetic architecture of ASDs is complex, requiring large samples to overcome heterogeneity. Here we broaden coverage and sample size relative to other studies of ASDs by using Affymetrix 10K SNP arrays and 1,168 families with at least two affected individuals, performing the largest linkage scan to date wh...

متن کامل

Mapping autism risk loci using genetic linkage and chromosomal rearrangements

Autism spectrum disorders (ASDs) are common, heritable neurodevelopmental conditions. The genetic architecture of ASDs is complex, requiring large samples to overcome heterogeneity. Here we broaden coverage and sample size relative to other studies of ASDs by using Affymetrix 10K SNP arrays and 1,168 families with at least two affected individuals, performing the largest linkage scan to date wh...

متن کامل

Chromosomal rearrangements and the genetics of reproductive barriers in mimulus (monkey flowers).

Chromosomal rearrangements may directly cause hybrid sterility and can facilitate speciation by preserving local adaptation in the face of gene flow. We used comparative linkage mapping with shared gene-based markers to identify potential chromosomal rearrangements between the sister monkeyflowers Mimulus lewisii and Mimulus cardinalis, which are textbook examples of ecological speciation. We t...

متن کامل

Comparative mapping between Arabidopsis thaliana and Brassica nigra indicates that Brassica genomes have evolved through extensive genome replication accompanied by chromosome fusions and frequent rearrangements.

Chromosome organization and evolution in the Brassicaceae family was studied using comparative linkage mapping. A total of 160 mapped Arabidopsis thaliana DNA fragments identified 284 homologous loci covering 751 cM in Brassica nigra. The data support that modern diploid Brassica species are descended from a hexaploid ancestor, and that the A. thaliana genome is similar in structure and complex...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:
  • Nature genetics

دوره 39 3  شماره 

صفحات  -

تاریخ انتشار 2007