Cell Protective Effect of Stress- Regulatory Peptide Urocortin in the Endogenous Adaptation Mechanisms of Myocardium
ثبت نشده
چکیده
منابع مشابه
CELL PROTECTIVE EFFECT OF STRESS-REGULATORY PEPTIDE UROCORTIN IN THE ENDOGENOUS ADAPTATION MECHANISMS OF MYOCARDIUM PhD Thesis By
s/Idézhető absztraktok: 1. Jancsó G., Borsiczky B., Szántó Z., Cserepes B., Karosi Z., Solymos A., Kiss K., Szeberényi J., Rőth E. :NFkB és AP-1 transzkripciós faktorok indukciójának dinamikája a prekondicionált miokardiumban Cardiologia Hungarica 2002. 32; 3. 193. 2. Jancsó G, Borsiczky B, Szántó Z, Cserepes B, Gasz B, Karosi Z, Solymos A, Kiss K, Szeberényi J, Rőth E. Nukleáris Faktor-kB és A...
متن کاملExploring new drugs for heart failure: the case of urocortin.
Over the past decade, clinical development of natriuretic peptide analogues as drug therapy for acute heart failure has sparked a wide range of interests to seek other endogenous vasoactive peptide systems that are operative as adaptive responders in human heart failure. With rapid advances in the molecular understanding of heart failure pathogenesis, several novel neurohormonal systems have be...
متن کاملEffect of six weeks low intensity interval training on expression of atrial and brain natriuretic peptide genes following myocardial infarction in rat
Background: Myocardial infarction (MI) is the irreversible cell death caused by ischemia in parts of myocardium. Atrial and Brain natriuretic peptides (ANP and BNP) are known as strong markers of myocardial infarction. This study was aimed to evaluate the effect of six weeks low intensity interval training on ANP and BNP genes expression in rats following myocardial infarction. Materials and m...
متن کاملMelatonin Protective Effects against Liver Ischemia/Reperfusion Injury
Hepatic ischemia-reperfusion (I/R) is a common phenomenon during liver surgery, transplantation, infection and trauma which results in damage and necrosis of the hepatic tissue through different pathways. Mechanisms involved in I/R damage are very intricate and cover several aspects. Several factors are involved in I/R-induced damages; briefly, decrease in sinusoidal perfusion and ATP generatio...
متن کاملUrocortin, but not urocortin II, protects cultured hippocampal neurons from oxidative and excitotoxic cell death via corticotropin-releasing hormone receptor type I.
Urocortin and urocortin II are members of the corticotropin-releasing hormone (CRH) family of neuropeptides that function to regulate stress responses. Two high-affinity G-protein-coupled receptors have been identified that bind CRH and/or urocortin I and II, designated CRHR1 and CRHR2, both of which are present in hippocampal regions of mammalian brain. The hippocampus plays an important role ...
متن کامل