Fukuyama-type Congenital Muscular Dystrophy and Abnormal Glycosylation of α-Dystroglycan

نویسندگان

  • Tatsushi Toda
  • Kazuhiro Kobayashi
  • Satoshi Takeda
  • Junko Sasaki
  • Hiroki Kurahashi
  • Hiroki Kano
  • Masaji Tachikawa
  • Fan Wang
  • Yoshitaka Nagai
  • Kiyomi Taniguchi
  • Mariko Taniguchi
  • Yoshihide Sunada
  • Toshio Terashima
  • Tamao Endo
  • Kiichiro Matsumura
چکیده

Fukuyama-type congenital muscular dystrophy (FCMD), Walker-Warburg syndrome (WWS), and muscle-eye-brain (MEB) disease are clinically similar autosomal recessive disorders characterized by congenital muscular dystrophy, lissencephaly, and eye anomalies. We identified the gene for FCMD and MEB, which encodes the fukutin protein and the protein O-linked mannose β1, 2-N-acetylglucosaminyltransferase (POMGnT1), respectively. α-dystroglycan is a key component of the dystrophin-glycoprotein-complex, providing a tight linkage between the cell and basement membranes by binding laminin via its carbohydrate residues. Recent studies have revealed that posttranslational modification of α-dystroglycan is associated with these congenital muscular dystrophies with brain malformations.

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Fukutin is prerequisite to ameliorate muscular dystrophic phenotype by myofiber-selective LARGE expression

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تاریخ انتشار 2003