Induction of HIV blocking anti - CCR 5 IgA in Peyers ’ s Patches 1 without histopathological alterations

نویسندگان

  • Claudia Pastori
  • Lorenzo Diomede
  • Assunta Venuti
  • Morgane Bomsel
  • Francesca Sanvito
  • Lucia Lopalco
چکیده

1 Division of Immunology, Transplantation and Infectious Diseases, San Raffaele 9 Scientific Institute, Milan, Italy 10 2 Molecular Biosensor and Imaging Center, Carnegie Mellon University, Pittsburgh, 11 PA, US 12 3 Inserm, U1016, Institut Cochin, Paris, France 13 4 CNRS UMR 8104, Paris, France 14 5 Entrée Muqueuse du VIH et Immunité Muqueuse Institut Cochin, Univ. Paris 15 Descartes Cité Paris Sorbonne, Paris, France 16 6 Pathology Department, Mouse Histopathology Unit, San Raffaele Scientific 17 Institute, Milan, Italy 18 # Corresponding author 19 8 These authors contributed equally 20 21 22 Running title: HIV protective CCR5 specific IgA 23 24 25 Corresponding author: [email protected] 26 27 28 29 JVI Accepts, published online ahead of print on 8 January 2014 J. Virol. doi:10.1128/JVI.03663-13 Copyright © 2014, American Society for Microbiology. All Rights Reserved.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Induction of HIV-blocking anti-CCR5 IgA in Peyers's patches without histopathological alterations.

UNLABELLED The chemokine receptor CCR5 is essential for HIV infection and is thus a potential target for vaccine development. However, because CCR5 is a host protein, generation of anti-CCR5 antibodies requires the breaking of immune tolerance and thus carries the risk of autoimmune responses. In this study, performed in mice, we compared 3 different immunogens representing surface domains of m...

متن کامل

Association between Anti-IgA Antibody and Development of Adverse Reactions in Immunodeficient Patients Under Immunoglobulin Therapy

Background: Mild to severe post-transfusion adverse reactions have been reported in a proportion of immunodeficient patients receiving intravenous immunoglobulin (IVIg). Anti-IgA antibody has been proposed to be associated with development of such reactions. Objective: To assess the association between anti-IgA levels of different isotypes and adverse reactions induced in immunodeficient patien...

متن کامل

Role of HIV-1 Tat and CC chemokine MIP-1alpha in the pathogenesis of HIV associated central nervous system disorders.

Two syndromes affecting cognitive and motor function in the setting of AIDS have been described as HIV encephalopathy (HIVE) and progressive multifocal leukoencephalopathy (PML). HIVE is characterized by the presence of microglial nodules with accompanying astrocytosis. PML is a fatal demyelinating disease of the white matter induced by the human papovavirus JCV which causes cytolytic destructi...

متن کامل

Alterations in salivary IgA levels in infectious and inflammatory disorders of upper respiratory tract

Background: The airway surfaces are one of the most common ways of entry of infectious agents. Oral cavity associated lymphoid tissues are inductive site of humoral immune responses in inflammatory and infectious disorders of the upper respiratory tract. These lymphoid tissues play important roles in the induction of salivary IgA. The impact of upper respiratory tract diseases on salivary IgA p...

متن کامل

Sphingosine 1-phosphate regulates the egress of IgA plasmablasts from Peyer's patches for intestinal IgA responses.

It is well established that Peyer's patches (PPs) are sites for the differentiation of IgA plasma cell precursors, but molecular and cellular mechanisms in their trafficking remain to be elucidated. In this study, we show that alterations in type 1 sphingosine 1-phosphate (S1P) receptor expression during B cell differentiation in the PPs control the emigration of IgA plasma cell precursors. Typ...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2014