Reduced Mid1 Expression and Delayed Neuromotor Development in daDREAM Transgenic Mice

نویسندگان

  • Mara Dierssen
  • Laura Fedrizzi
  • Rosa Gomez-Villafuertes
  • María Martinez de Lagran
  • Alfonso Gutierrez-Adan
  • Ignasi Sahún
  • Belen Pintado
  • Juan C. Oliveros
  • Xose M. Dopazo
  • Paz Gonzalez
  • Marisa Brini
  • Britt Mellström
  • Ernesto Carafoli
  • Jose R. Naranjo
چکیده

Downstream regulatory element antagonist modulator (DREAM) is a Ca(2+)-binding protein that binds DNA and represses transcription in a Ca(2+)-dependent manner. Previous work has shown a role for DREAM in cerebellar function regulating the expression of the sodium/calcium exchanger 3 (NCX3) in cerebellar granular neurons to control Ca(2+) homeostasis and survival of these neurons. To achieve a global view of the genes regulated by DREAM in the cerebellum, we performed a genome-wide analysis in transgenic cerebellum expressing a Ca(2+)-insensitive/CREB-independent dominant active mutant DREAM (daDREAM). Here we show that DREAM regulates the expression of the midline 1 (Mid1) gene early after birth. As a consequence, daDREAM mice exhibit a significant shortening of the rostro-caudal axis of the cerebellum and a delay in neuromotor development early after birth. Our results indicate a role for DREAM in cerebellar function.

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عنوان ژورنال:

دوره 5  شماره 

صفحات  -

تاریخ انتشار 2012