TGF-b Superfamily Cytokine MIC-1/GDF15 Is a Physiological Appetite and Body Weight Regulator

نویسندگان

  • Vicky Wang-Wei Tsai
  • Laurence Macia
  • Heiko Johnen
  • Tamara Kuffner
  • Rakesh Manadhar
  • Sebastian Beck Jørgensen
  • Ka Ki Michelle Lee-Ng
  • Hong Ping Zhang
  • Liyun Wu
  • Christopher Peter Marquis
  • Lele Jiang
  • Yasmin Husaini
  • Shu Lin
  • Herbert Herzog
  • David A. Brown
  • Amanda Sainsbury
  • Samuel N. Breit
چکیده

The TGF-b superfamily cytokine MIC-1/GDF15 circulates in all humans and when overproduced in cancer leads to anorexia/cachexia, by direct action on brain feeding centres. In these studies we have examined the role of physiologically relevant levels of MIC-1/GDF15 in the regulation of appetite, body weight and basal metabolic rate. MIC-1/GDF15 gene knockout mice (MIC-1(-/-)) weighed more and had increased adiposity, which was associated with increased spontaneous food intake. Female MIC-1(-/-) mice exhibited some additional alterations in reduced basal energy expenditure and physical activity, possibly owing to the associated decrease in total lean mass. Further, infusion of human recombinant MIC-1/GDF15 sufficient to raise serum levels in MIC-1(-/-) mice to within the normal human range reduced body weight and food intake. Taken together, our findings suggest that MIC-1/GDF15 is involved in the physiological regulation of appetite and energy storage.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2013