IFN-λ Inhibits Drug-Resistant HIV Infection of Macrophages

نویسندگان

  • Xu Wang
  • He Wang
  • Man-Qing Liu
  • Jie-Liang Li
  • Run-Hong Zhou
  • Yu Zhou
  • Yi-Zhong Wang
  • Wang Zhou
  • Wen-Zhe Ho
چکیده

Type III interferons (IFN-λs) have been demonstrated to inhibit a number of viruses, including HIV. Here, we further examined the anti-HIV effect of IFN-λs in macrophages. We found that IFN-λs synergistically enhanced anti-HIV activity of antiretrovirals [azidothymidine (AZT), efavirenz, indinavir, and enfuvirtide] in infected macrophages. Importantly, IFN-λs could suppress HIV infection of macrophages with the drug-resistant strains, including AZT-resistant virus (A012) and reverse transcriptase inhibitor-resistant virus (TC49). Mechanistically, IFN-λs were able to induce the expression of several important anti-HIV cellular factors, including myxovirus resistance 2 (Mx2), a newly identified HIV post-entry inhibitor and tetherin, a restriction factor that blocks HIV release from infected cells. These observations provide additional evidence to support the potential use of IFN-λs as therapeutics agents for the treatment of HIV infection.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2017