Large scale meta-analysis characterizes genetic architecture for common psoriasis associated variants

نویسندگان

  • Lam C Tsoi
  • Philip E Stuart
  • Chao Tian
  • Johann E Gudjonsson
  • Sayantan Das
  • Matthew Zawistowski
  • Eva Ellinghaus
  • Jonathan N Barker
  • Vinod Chandran
  • Nick Dand
  • Kristina Callis Duffin
  • Charlotta Enerbäck
  • Tõnu Esko
  • Andre Franke
  • Dafna D Gladman
  • Per Hoffmann
  • Külli Kingo
  • Sulev Kõks
  • Gerald G Krueger
  • Henry W Lim
  • Andres Metspalu
  • Ulrich Mrowietz
  • Sören Mucha
  • Proton Rahman
  • Andre Reis
  • Trilokraj Tejasvi
  • Richard Trembath
  • John J Voorhees
  • Stephan Weidinger
  • Michael Weichenthal
  • Xiaoquan Wen
  • Nicholas Eriksson
  • Hyun M Kang
  • David A Hinds
  • Rajan P Nair
  • Gonçalo R Abecasis
  • James T Elder
چکیده

Psoriasis is a complex disease of skin with a prevalence of about 2%. We conducted the largest meta-analysis of genome-wide association studies (GWAS) for psoriasis to date, including data from eight different Caucasian cohorts, with a combined effective sample size >39,000 individuals. We identified 16 additional psoriasis susceptibility loci achieving genome-wide significance, increasing the number of identified loci to 63 for European-origin individuals. Functional analysis highlighted the roles of interferon signalling and the NFκB cascade, and we showed that the psoriasis signals are enriched in regulatory elements from different T cells (CD8+ T-cells and CD4+ T-cells including TH0, TH1 and TH17). The identified loci explain ∼28% of the genetic heritability and generate a discriminatory genetic risk score (AUC=0.76 in our sample) that is significantly correlated with age at onset (p=2 × 10-89). This study provides a comprehensive layout for the genetic architecture of common variants for psoriasis.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2017