Helicobacter pylori induces AGS cell motility and elongation via independent signaling pathways.

نویسندگان

  • Stefan Moese
  • Matthias Selbach
  • Terry Kwok
  • Volker Brinkmann
  • Wolfgang König
  • Thomas F Meyer
  • Steffen Backert
چکیده

Helicobacter pylori induces motogenic and cytoskeletal responses in gastric epithelial cells. We demonstrate that these responses can be induced via independent signaling pathways that often occur in parallel. The cag pathogenicity island appears to be nonessential for induction of motility, whereas the elongation phenotype depends on translocation and phosphorylation of CagA.

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عنوان ژورنال:
  • Infection and immunity

دوره 72 6  شماره 

صفحات  -

تاریخ انتشار 2004