Fetal Hemoglobin Synthesis in Erythroid Cultures in Hereditary Persistence of Fetal Hemoglobin and @#{176}-Thalassemia By Rona

نویسندگان

  • S. Weinberg
  • Stylianos E. Antonarakis
  • Haig H. Kazazian
  • George J. Dover
  • Stuart H. Orkin
  • Alexandra L. Lenes
  • J. Matthew Schofield
  • Blanche P. Alter
چکیده

To determine whether hemoglobin regulation is normal in diseases affecting 1-globin gene expression. globin synthesis was examined in members of a family of a patient with hereditary persistence of fetal hemoglobin/fi#{176}-thalassemia (HPFH/$#{176}-thal). The HPFH defect is the Ghanian type II. with a deletion from t’$ to at least 20 kb 3’ to . The / #{176}-thaI gene has the haplotype II restriction enzyme pattern and has the 339 nonsense mutation. Erythroid cobnies from blood BFU-E were radiobabeled. and gbobin chains were separated by gel electrophoresis. Colonies from the $#{176}-thal heterozygote had non-a/a ratios more balanced than in the reticulocytes. Gamma synthesis was 1 1 % of

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Fetal Hemoglobin Synthesis in Erythroid Cultures in Hereditary Persistence of Fetal Hemoglobin and @#{176}-Thalassemia

To determine whether hemoglobin regulation is normal in diseases affecting 1-globin gene expression. globin synthesis was examined in members of a family of a patient with hereditary persistence of fetal hemoglobin/fi#{176}-thalassemia (HPFH/$#{176}-thal). The HPFH defect is the Ghanian type II. with a deletion from t’$ to at least 20 kb 3’ to . The / #{176}-thaI gene has the haplotype II restr...

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In vitro Induction of Fetal Hemoglobin in Erythroid Cells Derived from CD133 Cells by Transforming Growth Factor-b and Stem Cell Factor

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تاریخ انتشار 2005