Reconstruction of a Rare Palate Defect Originating from Extranodal Natural Killer/T-Cell Lymphoma

291 with the trauma. BCC is a very common disease entity. Therefore, based on the recognition of major skin trauma or wounds, patients spontaneously consider the BCC to have occurred in association with the trauma. However, there is no actual correlation between the two phenomena [3]. 2) The skin becomes more vulnerable to UV exposure because of the atrophy and decreased perfusion in the trauma-induced scar tissue. Thus, the nutritional supply is compromised, which eventually leads to a lack of ability to repair DNA damage [3]. 3) With chronic irritation to the damaged skin tissue, malignant transformation occurs. The damaged skin tissue releases toxins that may cause cellular mutation. Chronic wounds exposed over the longterm to these toxins become more vulnerable to malignant transformation [5]. 4) In cases of tumors arising from dense scar tissue, immunological privilege prevents lymphocyte infiltration. This interferes with the immune surveillance system. Thus, the tumor can protect itself from human defense mechanisms until its growth reaches a significant level [3]. 5) In the presence of trauma, the epidermal cells are implanted into the dermis. This triggers the occurrence of foreign body reactions in the dermis, alters the normal wound healing activity of the tissue, and renders the tissue more vulnerable to damage [5]. In cases of BCC arising from trauma or scar tissue, the degree of malignancy is not relatively higher than that due to other causes [1,3]. Therefore, standard treatment modalities are performed for the management of BCC. Our case highlights that BCC may also occur even after the trauma. Therefore, clinicians must rule out the possibility of malignant transformation when they examine patients with traumatic scars located on body surfaces that are vulnerable to sunlight exposure.