Recent evolution of clotting factor concentrates for hemophilia A and B. Transfusion Practices Committee.
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چکیده
TRANSFUSION1993—Vol. 33, No, 5FACTOR CONCENTRATES 1992433 12. Finlayson JS, Tankersley DL, Anti-HCV screening and plasmafractionation: the ease against (letter). Lancct 1990;335:1274-5.13. Pneumocystis carina pneumonia among persons with hemophiliaA . MMWR Morb Mortal Wkly Rep 1982;31:365-7.14. Krciss JK, Kitchen LW, Princc HE, Kasper CK, Essex M . Antibody to human T-lymphotropic virus type III in wives of hemophiliacs. Evidence for heterosexual transmission. Ann InternMed 1985;102:623-6.15. Ragni MV, Winkelstcin A , Kingslcy L , Spcro JA, Lewis JH. 1986update of HIV scroprevalence, seroconversion, AIDS incidence,and immunologic correlates of HIV infection in patients with hemophilia A and B. Blood 1987;70:786-90.16. Gocdcrt J J , Kcssler CM, Aledort LM, et al. A prospective studyof human immunodeficiency virus type 1 infection and the development of AIDS in subjects with hemophilia. N Engl J Med1989;321:1141-8.17. Risks associated with human parvovirus B19 infection. MMWRMorb Mortal Wkly Rep 1989;3.8:81-8,93-7.18. Frickhofen N, Abkowitz JL, Safford M, et ai. Persistent B19parvovirus infection in patients infected with human immunodeficiency virus type 1 (HIV-1): a treatable cause of anemia inAIDS. Ann Intern Med 1990;113:926-33.19. Mortimer PP, Luban NL, Ke lie her JF, Cohen BJ, Transmissionof scrum parvovirus-like virus by clotting-factor concentrates. Lancet1983;2:482-4.20. Morfini M, Longo G, Rossi Fcrrini P, cl al. Hypoplastic anemiain a hemophiliac first infused with a solvent/detergent treated factor VIII concentrate: the role of human B19 parvovirus (letter).Am J Hcmalol 1992;39:149-50.21. Azzi A , Ciappi S , Zakvrzewska K, Morfini M, Mariani G, Man-nucei PM. Human parvovirus B19 infection in hemophiliacs firstinfused with two high-purity, virally attenuated factor VIII concentrates. Am J Hematol 1992;39:228-30.22. Kasper CK. Thromboembolic complications. Thromb Diath Hac-morrh 1975;33:640-4.23. Lusher JM. Thrombogenicity associated with factor IX complexconcentrates. Semin Hematol 1991 ;2S(3 Suppl 6):3-5.24. Chavin SI, Siegel DM, Rosco T A Jr, Olson JP, Acute myocardialinfarction during treatment with an activated prothrombin complexconccntrate in a patient with factor VIII deficiency and a factorVIII inhibitor. Am J Med 1988;85:245-9.25. Mannucci PM, Bauer KA, Gringeri A, ct al. No activation of thecommon pathway of the coagulation cascade after a highly purified factor IX concentrate. Br J Haematol 1991;79:606-11.26. Menachc D, Behre HE, Orthner CL, et al. Coagulation factor IXconcentrate: method of preparation and assessment of potential invivo thrombogenicity in animal models. Blood 1984;64:1220-7.27. Kim HC, Matts L , Eisele J , Czachur M , Saidi P. Monoclonalantibody-purified factor IX—comparative thrombogenicity to prothrombin complex concentrate, Semin Hematol 1991;28(3 Suppl6): 15-9.28. Aronson DL, Finlayson JS. Direct and indirect effects of chronicpiasma protein infusion. In: Fratantoni JC, Aronson DL, cds.Unsolved therapeutic problems in hemophilia. US Dcpt of Health,Education, and Welfare publication No. (N1H) 77-1089, 1976:93-1 0 1 .29. Stein SF, Evalt BL, McDougal JS, ct al. A longitudinal study ofpatients wiih hemophilia: immunologic correlates of infection withHTLV-III/LAV and other viruses. Blood 1985;66:973-9,30. Cuthben RJ, Ludlam CA, Steel CM, Beatson D, Pcuthcrcr JF, Immunological studies in HIV seronegative haemophiliacs: relationships to blood product therapy. Br J Haemaiol 1992;80:364-9.31. Evans JA, Pasi KJ, Williams MD, Hill FG, Consistently normalCD4 + , C D 8 + levels in haemophilic boys only treated with avirally safe factor VIII concentrate (BPL 8Y). Br J Haematol1991;79:457-61.32. Thorpe R , Dilger P, Dawson NJ, Barrowciiffc T W . Inhibition ofintcrlcukin-2 secretion by factor VIII concentrates: a possible causeof immunosuppress ion in haemophi l iacs , Br J Haematol1989;71:387-91.33. Pasi KJ, Hill FG. In vitro and in vivo inhibition of monocytephagocytic function by factor VIII concentrates: correlation withconcentratc purity. Br J Haematol 1990;76:88-93,34. Madhok R , Smith J , Jenkins A , Lowe GD. T cell sensitization tofactor VIII in haemophilia A? Br J Haematol 1991;79:235-8.35. Eibl M M , Ahmad R, Wolf HM, Linnau Y , Gotz E , MannhalterJW. A component of factor VIII preparations which can be separated from factor VIII activity down modulates human monocytefunctions. Blood 1987;69:1153-60,36. Vcrmot-Desroches C , Rigal D, Blourde C , Bernaud J . Immunosuppressive property of a very high purity antihacmophilic preparation: a low molecular weight component inhibits an early stepof PHA induced cell activation. Br J Haematol 1992;80:370-7.37. Levine PH. Factor V1II:C purified from plasma via monoclonalantibodies: human studies, Semin Hematol I988;25(2 Suppl I):38-41.38. Cash JD . High potency factor VIII concentrates: value not proved?BMJ 1991;303:633-4.39. Goldsmith J M , Deutsche J , Tang M , Green D. CD4 cells inHIV-1 infected hemophiliacs: effect of factor VIII concentrates.Thromb Hacmost 1991;66:415-9.40. De Biasi R, Rocino A , Miraglia E, Mastrullo L, Quirino AA.The impact of a very high purity factor VIII concentrate on theimmune system of human immunodeficiency virus-infected hemophiliacs: a randomized, prospective, two-year comparison withan intermediate purity concentrate. Blood 1991;78:1919-22.41.. Mannucci PM, Gringeri A , de Biasi R, Baudo F, Morfini M,Ciavarclla N. Immune status of asymptomatic HIV-infcctcd hemophiliacs: randomized, prospective, two-year comparison oftreatment with a high-purity or an intermediate-purity factor VIIIconcentrate. Thromb Hacmost 1992;67:310-3.42. While G C II, McMillan C W , Kingdon HS, Shoemaker CB. Useof recombinant antihemophilic factor in the treatment of two patients with classic hemophilia. N Engl J Med 1989;320:166-70,43. Schwartz RS, Abildgaard CF, Aledort LM, ct al. Human recombinant DNA-derivcd antihemophilic factor (factor VIII) in thetreatment of hemophilia A . Recombinant Factor VIII Study Group.N Engl J Med 1990;323:1800-5.44. Horowitz B, Wicbe ME, Lippin A , Stryker MH. Inactivation ofviruses in labile blood derivatives. 1. Disruption of lipid-envclopcdviruses by tri(n-butyl)phosphate detergent combinations. Transfusion 1985;25:516-22.45. Horowitz B, Wiebc ME, Lippin A, Vandcrsandc J , Stryker MH,Inactivation of viruses in labile blood derivatives, 11, Physicalmethods. Transfusion 1985;25:523-7.46. Gompcrts E D . Procedures for the inactivation of viruses in clottingfactor concentrates. Am J Hematol 1986;23:295-305.47. Thomas DP. Reducing the risk of virus transmission by bloodproducts. Br J Haematol 1988;70:393-5.48. Ways to reduce the risk of transmission of viral infections byplasma and plasma products: a comparison of methods, their advantages and disadvantages. Vox Sang 1988;54:228-45.49. Brcttler DB, Levine PH. Factor conccntrates for treatment of hemophilia; which one to choose? Blood 1989;73:2067-73.50. Roberts HE, Eberst ME, The molecular biology of classic hemophilia and a review of factor VIII concentrates. In: Spivak JL,Bell WR, Ness PM, Qucsenberry PJ, Wiernik PH, cds. The yearbook of hematology. St Louis: Mosby Year Book, 1992:121-44, 51 . Heinrich D, Kotitschkc R, Bcrthold H, Clinical evaluation of thehepatitis safety of a bcta-propiolaelone/ultraviolet treated factorIX concentrate (PPSB). Thromb Res 1982;28:75-83.52. Schimpf K, Mannucci PM, Krcutz W , ct al. Absence of hepatitisafter treatment with a pasteurized factor VIII concentrate in patients with hemophilia and n o previous transfusions. N Enel JMed 1987;316:918-22.53. Colombo M, Mannucci PM, Carnclli V, Savidge GF, GazengclC, Schimpf K. Transmission of non-A, non-B hepatitis by heat-treated factor VIII concentrate. Lancct 1985;2:1-4.54. Medical Bulletin # 9 : Chapter Advisory # 1 2 . Medical and Scientific Advisory Council. New York: National Hemophilia Foundation, October 22, 1983.55. Skidmore SJ, Pasi KJ, Mawson SJ, Williams MD, Hill FG. Serological evidence that dry heating of clotting factor concentratesprevents transmission of non-A, non-B hepatitis. J Med Virol1990;30:50-2.
منابع مشابه
Factor concentrates for treatment of hemophilia: which one to choose?
U NTIL THE ADVENT of components that could more specifically replace specific missing clotting factors, patients with hemophilia were treated with whole blood and then plasma. Such patients had a shortened lifespan and were crippled with severe anthropathy; many died of hemonrhage. With the discovery in 1960 that factor VIII:C was concentrated in cryoprecipitate, more specific replacement for p...
متن کاملClotting factor concentrates given to prevent bleeding and bleeding-related complications in people with hemophilia A or B.
BACKGROUND People with severe hemophilia A or B, X-linked bleeding disorders due to decreased blood levels of coagulants, suffer recurrent bleeding into joints and soft tissues. Before clotting factor concentrates were available, most people with severe hemophilia developed crippling musculoskeletal deformities. Clotting factor concentrate prophylaxis aims to preserve joint function by converti...
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Factor IX concentrates were evaluated for their factor VII content and for the presence of activated factor VII through use of a coupled amidolytic assay insensitive to activated factor VII and a clotting assay sensitive to activated factor VII. The factor VII content of the concentrates studied (except for one concentrate purposely produced to exclude factor VII) varied between 33 and 621 U/vi...
متن کامل6 Factor VIII Concentrates, Factor VIII/von Willebrand Factor Concentrates, Factor IX Concentrates, Activated Prothrombin Complex Concentrates.
6.5 Range of Application, Dosage, Mode of Administration 6.5.1 General Information 6.5.2 Indications for Replacement Therapy Using Factor Concentrates 6.5.3 Dosage, Mode of Administration 6.5.3.1 Replacement in Children with Hemophilia A, B or von Willebrand Syndrome 6.5.3.2 Replacement in Adults with Hemophilia A, B or von Willebrand Syndrome 6.5.3.3 Indications and Recommended Doses for Treat...
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Background and Objectives: Hemophilia is a hereditary bleeding disorder, which CFC (clotting factor concentration) method is used for prevention and treatment of about 70% of these patients. This method can play an important role in the transmission of blood-borne viruses, such as hepatitis B and C. According to studies, more than 40% of patients with hemophilia have one of the hepatitis C, B, ...
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ورودعنوان ژورنال:
- Transfusion
دوره 33 5 شماره
صفحات -
تاریخ انتشار 1993