The metabolism of DL-1-deoxy-1-fluoroglycerol.
نویسندگان
چکیده
Clark, W. M. (1922). The Determination of Hydrogen Ions, p. 115. Dagley, S. & Sykes, J. (1956). Arch. Biochem. Biophys. 62, 338. Gomori, G. (1955). Methods in Enzymology, vol. 1, p. 138. Ed. by Kaplan, N. 0. & Colowick, S. P. New York: Academic Press Inc. Gunsalus, I. C., Galeener, C. C. & Stamer, J. R. (1955). Methods in Enzymology, vol. 2, p. 238. Ed. by Kaplan, N. 0. & Colowick, S. P. New York: Academic Press Inc. Happold, F. C. (1955). Ab8tr. 3rd Int. Congr. Biochem., Brussels, p. 32. Happold, F. C. & Struyvenberg, A. (1954). Biochem. J. 58, 379. Hellerman, L., Chinard, F. P. & Deitz, V. R. (1943). J. biol. Chem. 147, 443. Hopkins, F. C., Morgan, E. J. & Lutwak-Mann, C. (1938). Biochem. J. 32, 1829. Jakoby, W. B. & Bonner, D. M. (1953a). J. biol. Chem. 205, 699. Jakoby, W. B. & Bonner, D. M. (1953b). J. biol. Chem. 205, 709. Johnson, W. C. & Lindsey, A. J. (1939). Quoted by Vogel, A. I. (1951). Text-book of Quantitative Inorganic Analysis, p. 872. London: Longmans, Green and Co. Lowry, 0. H., Rosebrough, N. J., Farr, A. L. & Randall, R. J. (1951). J. biol. Chem. 193, 265. Mcllvaine, T. C. (1921). J. biol. Chem. 49, 183. Meister, A., Sobar, H. A. & Peterson, E. A. (1954). J. biol. Chem. 206, 89. Metzler, D. E., Ikawa, M. & Snell, E. E. (1954). J. Amer. chem. Soc. 76, 648. Sher, I. H. & Mallette, M. F. (1954). Arch. Biochem. Biophy8. 53, 354. Smith, E. L. (1948). J. biol. Chem. 173, 553. Sorensen, S. P. L. (1909). Biochem. Z. 22, 352. Spiegelman, S., Reiner, J. M. & Morgan, I. (1947). Arch. Biochem. 13, 113. Steensholt, G., Flikke, M. & Joner, P. E. (1955). Ab8tr. 3rd Int. Congr. Biochem., Brussels, p. 38.
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ورودعنوان ژورنال:
- The Biochemical journal
دوره 70 2 شماره
صفحات -
تاریخ انتشار 1958