[Frontiers in Bioscience 8, d117-134, January 1, 2003] 117 RETROVIRAL MUTATION RATES AND REVERSE TRANSCRIPTASE FIDELITY

نویسندگان

  • Evguenia S. Svarovskaia
  • Sara R. Cheslock
  • Wen-Hui Zhang
  • Wei-Shau Hu
  • Vinay K. Pathak
چکیده

1. Abstract 2. Importance of retroviral genetic variation 3. Historic developments in understanding retroviral genetic variation and RT fidelity 4. Factors that influence retroviral mutation rates 4.1. Reverse transcription 4.2. Other viral proteins 4.3. Nucleotide pools, DNA repair, and mammalian DNA polymerases 4.4. RNA transcription 4.5. RNA modification 4.6. Antiviral nucleoside analogs 5. In vivo fidelity assays and mutation rates 5.1. In vivo fidelity assays 5.2. In vivo mutation rates 6. In vitro fidelity assays and mutation rates 6.1. In vitro fidelity assays 6.2. In vitro mutation rates 7. Spectrum of mutations and their relative frequencies 8. Structural determinants of RT that influence fidelity 8.1. Structure of RT 8.2. Structural determinants of in vivo fidelity 8.3. Structural determinants of in vitro fidelity 8.4. The role of MLV RNase H primer grip and template-primer structure in fidelity 9. Future directions 10. Acknowledgements 11. References

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Retroviral recombination: review of genetic analyses.

Retroviruses package two copies of genomic RNA into one virion. One of the essential steps of replication is reverse transcription, in which the virally encoded enzyme reverse transcriptase (RT) uses the packaged RNA as a template to synthesize viral DNA. Because two copies of RNA are present in one virion, it is possible for RT to switch from one copy of the viral RNA to the other copy during ...

متن کامل

Mutation Rates and Intrinsic Fidelity of Retroviral Reverse Transcriptases

Retroviruses are RNA viruses that replicate through a DNA intermediate, in a process catalyzed by the viral reverse transcriptase (RT). Although cellular polymerases and host factors contribute to retroviral mutagenesis, the RT errors play a major role in retroviral mutation. RT mutations that affect the accuracy of the viral polymerase have been identified by in vitro analysis of the fidelity ...

متن کامل

HIV-1 mutagenesis during antiretroviral therapy: implications for successful drug treatment.

The evolution of antiretroviral drug resistance is a major problem in the treatment of human immunodeficiency virus type 1 (HIV-1) infection. Drug therapy failure is associated with accumulation of mutations and results in the development of drug resistance. Drugs targeted against reverse transcriptase (RT) as well as drug-resistant RT have been shown to increase HIV-1 mutation frequencies. Fur...

متن کامل

Comparison of Moloney murine leukemia virus mutation rate with the fidelity of its reverse transcriptase in vitro.

The role of Moloney murine leukemia virus (MoMLV) reverse transcriptase (RT) in the generation of base substitution mutations during retroviral replication was analyzed. To that effect, the in vitro fidelity of the MoMLV RT was compared to the rate of base substitution mutations occurring during the replication of an MoMLV-based retroviral vector. Using the vector in an amber reversion assay, t...

متن کامل

The high cost of fidelity.

The notoriously low fidelity of HIV-1 replication is largely responsible for the virus's rapid mutation rate, facilitating escape from immune or drug control. The error-prone activity of the viral reverse transcriptase (RT) is predicted to be the most influential mechanism for generating mutations. The low fidelity of RT has been successfully exploited by nucleoside and nucleotide analogue reve...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2002