Human Cancer Biology PI3K/AKT Signaling Is Essential for Communication between Tissue-Infiltrating Mast Cells, Macrophages, and Epithelial Cells in Colitis-Induced Cancer

نویسندگان

  • Mohammad W. Khan
  • Ali Keshavarzian
  • Elias Gounaris
  • Joshua E. Melson
  • Eric C. Cheon
  • Nichole R. Blatner
  • Zongmin E. Chen
  • Fu-Nien Tsai
  • Goo Lee
  • Hyunji Ryu
  • Terrence A. Barrett
  • David J. Bentrem
  • Philipp Beckhove
  • Khashayarsha Khazaie
چکیده

Purpose: Tounderstand signaling pathways that shape inflamed tissue andpredispose to cancer is critical for effective prevention and therapy for chronic inflammatory diseases.We have explored phosphoinositide 3-kinase (PI3K) activity in human inflammatory bowel diseases and mouse colitis models. Experimental Design: We conducted immunostaining of phosphorylated AKT (pAKT) and unbiased high-throughput image acquisition and quantitative analysis of samples of noninflamed normal colon, colitis, dysplasia, and colorectal cancer. Mechanistic insights were gained from ex vivo studies of cell interactions, the piroxicam/IL-10 / mouse model of progressive colitis, and use of the PI3K inhibitor

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PI3K/AKT signaling is essential for communication between tissue-infiltrating mast cells, macrophages, and epithelial cells in colitis-induced cancer.

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تاریخ انتشار 2013