Structural insights into the DNA-binding specificity of E2F family transcription factors

نویسندگان

  • Ekaterina Morgunova
  • Yimeng Yin
  • Arttu Jolma
  • Kashyap Dave
  • Bernhard Schmierer
  • Alexander Popov
  • Nadejda Eremina
  • Lennart Nilsson
  • Jussi Taipale
چکیده

The mammalian cell cycle is controlled by the E2F family of transcription factors. Typical E2Fs bind to DNA as heterodimers with the related dimerization partner (DP) proteins, whereas the atypical E2Fs, E2F7 and E2F8 contain two DNA-binding domains (DBDs) and act as repressors. To understand the mechanism of repression, we have resolved the structure of E2F8 in complex with DNA at atomic resolution. We find that the first and second DBDs of E2F8 resemble the DBDs of typical E2F and DP proteins, respectively. Using molecular dynamics simulations, biochemical affinity measurements and chromatin immunoprecipitation, we further show that both atypical and typical E2Fs bind to similar DNA sequences in vitro and in vivo. Our results represent the first crystal structure of an E2F protein with two DBDs, and reveal the mechanism by which atypical E2Fs can repress canonical E2F target genes and exert their negative influence on cell cycle progression.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Dna - Binding Specificity of the E 2 F Gene Family , and Cloning and Characterization of a Novel Family

The E2F transcription factors have been implicated in the regulation of cell cycle progression and apoptosis. The E2F proteins are differentially regulated, and have distinct functions in addition to any shared ones. This work describes the identification of a novel E2F protein, E2F-6. Unlike the previously characterized E2F proteins, E2F-6 lacks a transcriptional activation domain and does not...

متن کامل

Covariation between homeodomain transcription factors and the shape of their DNA binding sites

Protein-DNA recognition is a critical component of gene regulatory processes but the underlying molecular mechanisms are not yet completely understood. Whereas the DNA binding preferences of transcription factors (TFs) are commonly described using nucleotide sequences, the 3D DNA structure is recognized by proteins and is crucial for achieving binding specificity. However, the ability to analyz...

متن کامل

Post-Transcriptional Regulation of E2F Transcription Factors: Fine-Tuning DNA Repair, Cell Cycle Progression and Survival in Development & Disease

Cells are continually exposed to genotoxic stresses. Upon DNA damage, the cell activates a coordinated and complex series of responses (Levitt and Hickson, 2002). Multiple factors are implicated in each of these responses. Recently, it has become apparent that various transcription factors play important roles in cellular responses to genotoxic stress. In particular, E2F transcription factors a...

متن کامل

The use of transient chromatin immunoprecipitation assays to test models for E2F1-specific transcriptional activation.

The E2F family of transcription factors regulates the expression of genes involved in cell cycle progression, DNA synthesis, repair, and recombination, and a variety of other cellular processes. Although E2F proteins are often redundant in function, specificity of binding and activity can occur. For example, E2F1, but not other E2F family members, was shown previously to bind the murine carboxy...

متن کامل

Characterization of E2F8, a novel E2F-like cell-cycle regulated repressor of E2F-activated transcription

The E2F family of transcription factors are downstream effectors of the retinoblastoma protein, pRB, pathway and are essential for the timely regulation of genes necessary for cell-cycle progression. Here we describe the characterization of human and murine E2F8, a new member of the E2F family. Sequence analysis of E2F8 predicts the presence of two distinct E2F-related DNA binding domains sugge...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2015