Claudication without risk factors: a case of popliteal entrapment syndrome.

To cite: Shalabi ST, Hussain N, Shalabi HT, et al. BMJ Case Rep Published online: [please include Day Month Year] doi:10.1136/ bcr-2013-202199 DESCRIPTION A 54-year-old man presented with claudication in his left calf with a walking distance of 200 m. There was no history of hypertension, hyperlipidaemia, thrombophilia, smoking or any risk factors associated with atherosclerosis. Doppler scan demonstrated occlusion from the distal superficial femoral artery (SFA) to the proximal popliteal artery. A lower limb MR angiography supported this and showed significant collateralisation (figure 1). He underwent CT angioplasty with immediate symptomatic relief. The relief was temporary, as 2 months later he presented with a recurrence of symptoms. The case was atypical as there were no significant risk factors and while the lesion was attributed to embolus, no cardiac source could be found. Therefore, the possibility of popliteal artery entrapment syndrome (PAES) was considered. A further CT scan in the neutral and plantar flexed positions was able to demonstrate a short tight stenosis in the popliteal artery which was lying abnormally; medial rather than lateral to the medial head of gastrocnemius (figure 2). This correlated with type 2 PAES. In theatre the patient was placed in the prone position, and an S-shaped incision was made exposing the popliteal fossa. The medial gastrocnemius ran from medial to lateral compressing the popliteal artery behind the knee in a sling fashion (figure 3). The sling was divided allowing the artery to lie free (figure 4). The SFA above the compression was hard to touch.