Lipase Activity With Increasing Intra-abdominal Fat in Women A Hepatic Lipase Gene Promoter Polymorphism Attenuates the Increase in Hepatic

نویسندگان

  • John D. Brunzell
  • Molly C. Carr
  • John E. Hokanson
  • Samir S. Deeb
  • Jonathan Q. Purnell
  • Ellen S. Mitchell
چکیده

High hepatic lipase (HL) activity is associated with an atherogenic lipoprotein profile of small, dense LDL particles and lower HDL2-C. Intra-abdominal fat (IAF) is positively associated with HL activity. A hepatic lipase gene (LIPC) promoter variant (G3A) is associated with lower HL activity, higher HDL2-C, and less dense LDL particles. To determine whether the LIPC promoter polymorphism acts independently of IAF to regulate HL, 57 healthy, premenopausal women were studied. The LIPC promoter A allele was associated with significantly lower HL activity (GA/AA5104634 versus GG5145657 nmoles z mL z min, P50.009). IAF was positively correlated with HL activity (r50.431, P,0.001). Multivariate analysis revealed a strong relationship between both the LIPC promoter genotype (P50.001) and IAF (P,0.001) with HL activity. The relationship between IAF and HL activity for carriers and noncarriers of the A allele was curvilinear with the carriers having a lower apparent maximum level of plasma HL activity compared with noncarriers (138 versus 218 nmoles z mL z min, P,0.001). In addition, the LIPC A allele was associated with a significantly higher HDL2-C (GA/AA51667 versus GG51165 mg/dL, P50.003). We conclude that the LIPC promoter A allele attenuates the increase in HL activity due to IAF in premenopausal women. (Arterioscler Thromb Vasc Biol. 1999;19:2701-2707.)

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A hepatic lipase gene promoter polymorphism attenuates the increase in hepatic lipase activity with increasing intra-abdominal fat in women.

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تاریخ انتشار 1999