Identification of FAM3D as a novel endogenous chemotaxis agonist for the FPRs (formyl peptide receptors)

نویسندگان

  • Xinjian Peng
  • Enquan Xu
  • Weiwei Liang
  • Xiaolei Pei
  • Dixin Chen
  • Danfeng Zheng
  • Yang Zhang
  • Can Zheng
  • Pingzhang Wang
  • Shaoping She
  • Yan Zhang
  • Jing Ma
  • Xiaoning Mo
  • Yingmei Zhang
  • Dalong Ma
  • Ying Wang
چکیده

Department of Immunology, School of Basic Medical Sciences, and Key Laboratory of Medical Immunology of Ministry of Health, Peking University Health Science Center, Beijing 100191, China. Center for Human Disease Genomics, Peking University, Beijing 100191, China. *These authors contributed equally to this paper. Correspondence author: Ying Wang, 38 Xueyuan Road, Beijing 100191, China. Tel.: +86-10-82802846-5032; Fax: +86-10-82801149; E-mail: [email protected]

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منابع مشابه

International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the formyl peptide receptor (FPR) family.

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The Formyl Peptide Receptors: Diversity of Ligands and Mechanism for Recognition.

The formyl peptide receptors (FPRs) are G protein-coupled receptors that transduce chemotactic signals in phagocytes and mediate host-defense as well as inflammatory responses including cell adhesion, directed migration, granule release and superoxide production. In recent years, the cellular distribution and biological functions of FPRs have expanded to include additional roles in homeostasis ...

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N-Formyl peptide receptors (FPRs: FPR1, FPR2, and FPR3) are G protein-coupled receptors that play key roles in modulating immune cells. FPRs represent potentially important therapeutic targets for the development of drugs that could enhance endogenous anti-inflammation systems associated with various pathologies, thereby reducing the progression of inflammatory conditions. Previously, we identi...

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N-formyl peptide receptor (FPR1) and N-formyl peptide receptor-like 1 (FPRL1, now known as FPR2) are G protein-coupled receptors involved in host defense and sensing cellular dysfunction. Because of the potential for FPR1/FPR2 as a therapeutic target, our recent high-throughput screening efforts have focused on the identification of unique nonpeptide agonists of FPR1/FPR2. In the present studie...

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تاریخ انتشار 2016