MYELOID NEOPLASIA Frequent CBL mutations associated with 11q acquired uniparental disomy in myeloproliferative neoplasms

نویسندگان

  • Francis H. Grand
  • Claire E. Hidalgo-Curtis
  • Thomas Ernst
  • Katerina Zoi
  • Christine Zoi
  • Carolann McGuire
  • Sebastian Kreil
  • Amy Jones
  • Joannah Score
  • Georgia Metzgeroth
  • David Oscier
  • Andrew Hall
  • Christian Brandts
  • Hubert Serve
  • Andreas Reiter
  • Andrew J. Chase
  • Nicholas C. P. Cross
چکیده

1Wessex Regional Genetics Laboratory, Salisbury, and Human Genetics Division, School of Medicine, University of Southampton, Southampton, United Kingdom; 2Haematology Research Laboratory, Biomedical Research Foundation, Academy of Athens, Athens, Greece; 3Inflammation, Infection and Repair Division, School of Medicine, University of Southampton, Southampton, United Kingdom; 4III Medizinische Universitätsklinik, Fakultät für Klinische Medizin Mannheim der Universität Heidelberg, Mannheim, Germany; 5Department of Haematology, Royal Bournemouth Hospital, Bournemouth, United Kingdom; 6Northern Institute for Cancer Research, Newcastle upon Tyne, United Kingdom; and 7Department of Medicine, Hematology & Oncology, University of Frankfurt, Frankfurt, Germany

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Frequent CBL mutations associated with 11q acquired uniparental disomy in myeloproliferative neoplasms.

Recent evidence has demonstrated that acquired uniparental disomy (aUPD) is a novel mechanism by which pathogenetic mutations in cancer may be reduced to homozygosity. To help identify novel mutations in myeloproliferative neoplasms (MPNs), we performed a genome-wide single nucleotide polymorphism (SNP) screen to identify aUPD in 58 patients with atypical chronic myeloid leukemia (aCML; n = 30)...

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تاریخ انتشار 2009