Mutations in ribosomal protein L3 are associated with oxazolidinone resistance in staphylococci of clinical origin.

نویسندگان

  • Jeffrey B Locke
  • Mark Hilgers
  • Karen Joy Shaw
چکیده

Following recent reports of ribosomal protein L3 mutations in laboratory-derived linezolid-resistant (LZD(r)) Staphylococcus aureus, we investigated whether similar mutations were present in LZD(r) staphylococci of clinical origin. Sequence analysis of a variety of LZD(r) isolates revealed two L3 mutations, DeltaSer145 (S. aureus NRS127) and Ala157Arg (Staphylococcus epidermidis 1653059), both occurring proximal to the oxazolidinone binding site in the peptidyl transferase center. The oxazolidinone torezolid maintained a >or=8-fold potency advantage over linezolid for both strains.

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عنوان ژورنال:
  • Antimicrobial agents and chemotherapy

دوره 53 12  شماره 

صفحات  -

تاریخ انتشار 2009